Clostridium Perfringens Essay

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Abeer Tadrus and Hadjer Sahraoui BIO-325-01
Dr. McLaughlin 3/20/2014
Clostridium perfringens
Clostridium perfringens is a gram-positive spore-forming bacillus involved in foodborne illness and wound infection. It is an obligate anaerobe and the only member of the genus Clostridium that is non-motile. This microorganism is normally present in soil and decaying vegetation and is an inhabitant of animal and human intestines. According to the Centers for Disease Control and Prevention, C. perfringens is one of the most common sources of foodborne illness in the United States, being the cause of an estimated 1 million cases each year. However, it is also prevalent worldwide. This bacterium has the shortest reported generation time of any organism at 6.3 minutes in thioglycollate medium, making it particularly virulent after initial inoculation.
Some strains of this microorganism produce C. perfringens enterotoxin (CPE), a group of toxins that cause a variety of adverse effects in the host. Strains of C. perfringens are classified as 5 biotypes, A – E, depending on the production of four major enterotoxins (α, β, ε and ι). In addition, strains of C. perfringens may also produce a number of other toxins including neuraminidase, hyaluronidase, and collagenase. For example, α-toxin, produced by C. perfringens type A, is primarily responsible for the production of gas gangrene. However, only roughly 5% of C. perfringens carry the CPE gene that codes for the production of these toxins. CPE is inactivated at 74oC.
Infection with C. perfringens as a food-borne illness is acquired from consumption of food or water that has come in contact with the bacterium. Some possibilities include unwashed fruits and vegetables, undercooked me...

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...50% of healthy humans. According to the results of this study, 52% of the healthy controls (n = 31) were carriers of C. perfringens type A. This study showed that 23% of C. perfringens type A is present in individuals with MS. In other words, Clostridium perfringens type A, which is commensal to humans, was present in roughly half of healthy the healthy controls as opposed to 23% in patients with MS. Also, 10% of MS patients and 1% of controls possessed ETX specific antibodies.
For CSF, 6 of 62 MS patients, and 1 of 40 controls, were positive for ETX immunoreactivity. For the analysis of sera, 6 of 56 MS patients, and 0 of 60 controls were seroreactive to ETX.
According to this experiment, it is clear that C. perfringens has specificity toward CNS epithelial cells and white matter. It is further studied that EXT binds to retinal vessels and myelin in the cranium.

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