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Cardiovascular disease, also known as heart disease, is a term used for diseases involving the heart, arteries, capillaries and veins. The problems associated with cardiovascular disease are often a result of atherosclerosis. Atherosclerosis is caused by a buildup of plaque in artery walls, which disrupts blood flow through the arteries (American Heart Association, 2011). Cardiovascular disease causes a variety of conditions including heart attacks, ischemic stroke, heart failure, coronary artery disease, arrhythmias and heart valve problems (American Heart Association, 2011). These conditions lead to serious health related issues for individuals, including death. Every year there are around 600,000 individuals in the United States that die from cardiovascular disease, making heart disease the leading cause of death in both men and women (CDC, 2014). Although there are ways to decrease the risk of heart disease, the rate has been consistently increasing over the years, costing America billions of dollars in health care services annually. Contributing factors to the rise in heart disease includes the rate of obesity and a lack of physical activity (Dhaliwal, Welborn & Howat, 2013; Poirier, Giles, Bray, Hong, Pi-Sunyer & Eckel, 2006). These articles provide research that answers the question of how obesity and physical activity are linked to cardiovascular disease.
Obesity and Cardiovascular Disease
Along with an increase in cardiovascular disease, the rate of obesity is rising as well. Obesity is linked to a number of other diseases, known as comorbidities, including cardiovascular disease. Because of the adjustments the body must make to function in severely overweight individuals, cardiac structure must adapt to the adipose tiss...
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...al conditions of cardiovascular disease that have been linked to obesity, including strokes, coronary artery disease, congestive heart failure and arrhythmias. All of these conditions can be improved or prevented by the individual losing ten percent of their body weight. Physical activity also plays a crucial role in cardiovascular disease. Studies have shown that the greater the amount of physical activity, the less the chance for developing cardiovascular disease, even when other factors, such as Framingham’s scale, are accounted for. Regarding future research, a study further researching genetic and environmental links to cardiovascular disease would provide helpful information. Also, research studying the effects of physical activity after the onset of cardiovascular disease could be beneficial to determine if physical activity can improve patients’ conditions.
Hyperlipidemia or increased cholesterol builds up causing narrowing in the arteries, which reduces oxygen rich blood flow to the heart and tissues, also increasing the risk for myocardial infarction. Obesity increases the risk of developing diabetes, high blood pressure, and hyperlipidemia, increasing the risk of atherosclerosis, and therefore increasing
Thompson, P. D., Buchner, D., Pina, I. L., Balady, G. J., Williams, M. A., Marcus, B. H., ... Wenger, N. K. (2003). Exercise in the prevention and treatment of atherosclerotic cardiovascular disease: a statement from the Council on Clinical Cardiology. Journal of the American Heart Association, 3110-3116. http://dx.doi.org/doi: 10.1161/01.CIR.0000075572.40158.77
Cardiomyopathy, by definition, means the weakening of the heart muscle. The heart is operated by a striated muscle that relies on the autonomic nervous system to function. Cardiomyopathy is diagnosed in four different ways based on what caused the illness and exactly what part of the heart is weakened. The four main types of cardiomyopathy are dilated cardiomyopathy, hypertrophic cardiomyopathy, restrictive cardiomyopathy, and arrhythmogenic right ventricular dysplasia. One other category of cardiomyopathy that is diagnosed is “unclassified cardiomyopathy.” Unclassified cardiomyopathy is the weakening of the heart that does not fit into the main four categories.
It was to this respect that, the search could detect ‘’hypertension’’ as the leading risk factor for heart disease. And this preceded three quarters of heart failures cases as compared to coronary artery disease, which led to most heart failures in less than 40% of the cases. Also, an increase in left ventricular end-diastolic diameter became a mirror to the Framingham study as incident heart diseases in the individuals who are free from myocardial infarction. Although studies have shown that, the manifestation of heart failures can be present without the left ventricular systolic dysfunction, other risk factors could lead to that. Also, they (Framingham study) were able to detect ‘’too much of cholesterol’’ as a link to cardiovascular diseases. Moreover, research believed that has elevated among certain heart diseases such as coronary heart often leads to stroke, too high blood pressure among numerous patients. Having said that, the search discovered ‘’obesity’’ also as a concomitantly with hypertension which elevates lipids and diabetes versus questions on smoking behavior. Having said that, these risk factors are believed to have attributed to heart diseases. Therefore, it became a national concern to the general US population and that of the fourth director of Framingham heart study, William Castelli
Harvard Medical Group “Best medicine: The science of exercise shows benefits beyond weight …..loss.” Harvard Heart Letter. 23(11) (2013) 6
Why do we need to exercise? With out exercise many of our bodies arteries in the cardiovascular system can become clogged and bring on much unwanted cardiovascular diseases. Exercising regularly helps us maintain a healthy weight if already fi...
Being overweight or obese are risk factors for many chronic health conditions such as heart disease, diabetes, and … cancers.”
Coronary artery disease (CAD) is caused by reduced blood flow in the coronary arteries. This subsequently leads to reduced oxygenation to the myocardium, resulting in transient ischemia or angina. CAD may cause permanent damage to myocardial cells or infarction. The left ventricle of the heart is most susceptible to CAD.
“Obesity Information.” American Heart Association Obesity Information. American heart Association, 27 February 2014. Web. 04 Apr. 2014
(Slide 2) What is Cardiomyopathy? If we break down the word we can see “Cardio” which means of the heart, “myo” which means muscle, and “pathy” which means disease, therefore cardiomyopathies are diseases of the heart muscle. (Slide 3) There are 3 main types of cardiomyopathies; hypertrophic, dilated, and restrictive. I will only be discussing dilated cardiomyopathy, which is characterized by the enlargement of the hearts chambers with impaired systolic function. It is estimated that as many as 1 of 500 adults may have this condition. Dilated cardiomyopathy is more common in blacks than in whites and in males than in females. It is the most common form of cardiomyopathy in children and it can occur at any age (CDC).
Atherosclerosis is a disease that occurs when arteries become blocked, inflamed, or hardened. As a result of this, blood cannot easily pass through the artery, and blood pressure increases. Many people suffer from atherosclerosis as they age, but young people can be affected by atherosclerosis also. There are many preventative steps that can be taken to decrease the risk of atherosclerosis; however, if atherosclerosis does develop in the arteries, medications can be given to help the individual receive adequate blood flow to important tissues. Atherosclerosis is a very serious condition that requires medical attention and a change in life style because it is a precursor to many dangerous and potentially fatal diseases.
Millions of Americans and people worldwide are overweight or obese. Obesity develops when “calories consumed exceeds calories expended” (“Obesity and Genetics”). “Obesity rates have more than doubled in adults and children since the 1970’s,” and in the present day it is estimated that “two – thirds of U.S. adults are overweight or obese” (Ogden). Being overweight or obese highly increases the risk of deadly health problems, therefore this statistic states that the majority of the United States population is at risk of obtaining life–threatening diseases. Around forty years ago obesity would not effect this abundant number of people; however today’s society consumes more fast food in addition to spending most of their hours doing sedentary activities (Green). There are now many causes of obesity such as environment, genetics, bad habits, culture and economic level. Obesity has many negative impacts on the human body. It can very likely cause diabetes, joint pain, sleeping problems, depression, and many hazardous diseases (“Explore Overweight”). In contrast to this, there are some possible solutions to obesity such as physical activity, dieting, and surgery. Obesity is a widespread epidemic that unfavorably affects the body, but with exercise fused with dieting the disease could be kept to a minimum.
Oxygen was first admitted to the client with chest pain over 100 years ago (Metcalfe, 2011). Chest pain is a large bracket that can contain many different conditions, but for the purpose of this analysis it is focused manly upon a myocardial infarction. A myocardial infarction is mainly referred to as a heart attack, and occurs when one or more coronary arteries leading to the heart reduce or completely stop blood flow (Tuipulotu, 2013 ). Administering high concentrations of oxygen to patients with chest pain is now embedded in guidelines, protocols and care pathways, even with a lack of clear supporting evidence (Nicholson, 2004 ). High concentration of oxygen means that up to 60% is administered (Knott, 2012). More recent research has suggested that the use of oxygen in this scenario is unnecessary and can lead to unwanted side effects, especially in normoxic cardiac patients (Moradkham & Sinoway, 2010 ). The aim of this comparative analysis is to dismantle and understand both the benefits and risks of the commonly known practice of administration of oxygen to the client with chest pain. Through completing this analysis using recent and appropriate evidence a more improved practice can be given and understood.
Atherosclerosis, one of the leading causes of death in the country, is a condition in the arteries that is characterized by the deposition of plaque on the arterial walls. While the exact cause of atherosclerosis is unknown, it is a slow, complex disease that begins developing during childhood, and progresses faster with age.1 Certain factors such as smoking, obesity, high cholesterol, high blood pressure, and increased concentrations of sugar in the blood (or diabetes), play a role in the development of atherosclerosis by damaging the endothelium and inner layers of the arteries.1 Plaque begins to build up and harden in the areas where the inner layers of the arteries are damaged.1 These areas of plaque potentially rupture or burst, which encourages platelets to attack to the site of the injury and clump there, forming blood clots.1 These blood clots combined with plaque narrow arteries even more, which further limits the flow and delivery of oxygenated blood to the rest of the body.1 Atherosclerosis can ultimately cause a heart attack (blood flow to the heart is blocked), a stroke (blood flow to the brain is blocked), or angina (chest pain) depending on the artery damaged.1
Coronary heart disease (CHD) is a group of syndromes that are caused from plaque built up inside the coronary arteries1. CHD is the most common cause of death worldwide, with more than 7 million deaths per year2. As a significant public health issue, CHD has been intensively studied for its risk prediction3–8. The initial models have been developed with risk factors such as age, blood pressure, cigarette smoking, total cholesterol, high density lipoprotein cholesterol, and diabetes status3. To improve prediction of CHD beyond traditional risk factors, there were endeavors to add emerging risk factors such as C-reactive protein8–10 and risk scores using candidate genes11,12. Advances in genomic technologies led to identification of novel CHD susceptibility genes from large-scale genome-wide association studies (GWAS) in various ethnicities 13–39.