Rheumatoid Arthritis Case Study

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Rheumatoid Arthritis
Insert your name in the header. Type your answers in RED, save your final copy in Word (.doc or .docx). Send as an attachment to your clinical instructor and cc Miss Williams. In order to receive full attendance credit, we MUST receive your assignment by: 0630 9/27/16 and med cards by 0800 9/28/16.
1. What 3 questions the nurse should ask when assessing a client’s ROM with RA?
a. Is there pain on motion?
b. Is there limitation of motion? Can patient to active or passive motion?
c. Assess muscle tone, strength, coordination
d. Does the patient need assistive devices to perform ADL? (recommend occupational therapist)
e. Idk this one.. google it

2. What are 3 important considerations for the nurse when overseeing the care …show more content…

Provide answers to the following, you may consult evidence based resources to provide answers to the following (cite your source in APA format):
Explain what the fallowing pathophysiological changes to AUTOIMMUNE REACTION
(Please write in your own words and cite your reference)
CHANGE WHAT DOES IT MEAN
Antigens to T cells T cells are used to find infected cells that are tagged with a specific antigen. Once the antigen is in contact, the T cells will destroy the cell; this results in cell mediated immunity. (ahn pg 740)
Inflammatory process The process of inflammation helps the body get rid of dead tissue and pathogens in order to repair body tissues and cells. When the inflammatory response is triggered (by chemical and physical agents or microorganisms), the site injured is brought nutrients, products of blood, and fluid. The signs of inflammation include loss of function, edema, pain, erythema, pus drainage, and heat. (FON pg 125 or 289)

Increased capillary permeability In inflamed tissues, the capillaries will have increased permeability. This causes edema at the injured site due to the increased blood flow (to carry nutrients and white blood cells to repair the injury). …show more content…

This can lead to pain, swelling, inflammation, and damage to the joints affected. http://www.arthritis-health.com/types/rheumatoid/what-pannus T- and B cell proliferation Angiogenesis in synovial lining T and B cells are some of the main immune cells for synovial membrane inflammation. T cells proliferate in the synovial membrane in response to a specific antigen (ex: cartilage autoantigen) (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3897168/). B cells produce cytokines and chemokines to promote angiogenesis and leukocyte infiltration in the joints (https://www.ncbi.nlm.nih.gov/pubmed/15180890) . Angiogenesis in the synovial lining can be a key factor in treating arthritis

8. Make medication cards for (due on 9/28 at 0800): glucosamine, celecoxib, methotrexate, leflunomide, meloxicam
9. Formulate a Nursing Care plan (3-part statement incl R/T and WEB) for a client with RA
With one short-term/one long-term goal, at least 4 nursing interventions with scientific

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