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Abstract essay sleep disorders
Abstract essay sleep disorders
Abstract essay sleep disorders
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Thank you for referring Jim Mitzas, a 45-year-old team leader who works for Energy Australia in an office environment. Jim is a non-smoker and seldom consumes alcohol. There is a longstanding history of Crohn’s disease that has been quite stable with mesalazine. Other current medications are Pariet, Panadeine Forte and Brufen. Regarding his sleep, Jim usually retires to bed at around 11.30pm with what he perceives to be prolonged sleep onset latency of around one hour. He often has trouble finding a comfortable position and reports an overactive mind. There is a history of snoring, witnessed apnoea, nocturia, sleep fragmentation and when he wakes at 5.30am, he consistently feels unrefreshed. Jim is also been noted to sit up at the …show more content…
Chest auscultation was clear and two heart sounds were audible with nil else. Prior to this appointment, Jim has undergone a home-based diagnostic sleep study through the Austin. This has revealed short sleep onset latency of 3.5 minutes with reduced sleep efficiency. There is severe obstructive sleep apnoea with an Apnoea-Hypopnoea Index of 84 events/hr. There was significant oxygen desaturation during the night with more than 50% time of total sleep time spent with a saturation of less than 88%. There were no notable periodic limb movements. I have discussed the findings with Jim and given the severity of his sleep apnoea, CPAP is certainly indicated. He will commence treatment at home, initially using an auto-set machine followed by a fixed pressure device. I will review him again in a month to assess his progress. He will notify VicRoads of the sleep apnoea diagnosis. I do agree that in due course undergoing a tonsillectomy and septoplasty will likely improve his sleep apnoea and allow use of lower CPAP pressures. Hopefully, Jim will also be able to make an attempt at weight-loss with improved energy
The case study of “What should we do with Jim?” has been read and a set amount of questions has been asked about the reading, which will be answered by the following:
...re shortness of breath during night presenting itself in episodes, usually where the individual suddenly wakes up quite panicked and anxious it known as Paroxysmal nocturnal dyspnoea (Bozkurt). Paroxysmal nocturnal dyspnea is caused by abnormal blood or fluid in the lungs, usually the individual stands up the fluid moves down that is why when Mr Smith has an paroxysmal nocturnal dyspnoea episode he is relieved by sitting on the side of his bed, in doing so the fluid moves down away from his lungs. If Mr Smith does suffer from pulmonary hypertension and if it were to worsen his paroxysmal nocturnal dyspnea would not be relieved by standing up or sitting down on the side of his bed as the pulmonary hypertension eventually weaken the left ventricle of his heart which in affect would not be able to pump out the fluid resulting in continued shortness of breath (T. doug).
Unfortunately the referral letter was not available at the time of review however I gather the reason for referral is that Kori has been suffering with sleep onset insomnia and unrefreshing sleep. He usually retires to bed at 10.30pm but will generally not be able to sleep for at least an hour. On occasions he will then listen to some soft music and then make another attempt to fall asleep. Often he does not fall asleep until 1.30am to 2.00am. There is no history or snoring, witness apnoeas or nocturnal choking episodes but Kori's sleep is fragmented but there is no sleep maintenance insomnia. He reports general restless but no specific restless legs symptoms as such. When he awakes at 7.00am, he consistently feels unfreshed and he is aware of some daytime somnolence although the Epworth Sleepiness score is normal at 7/24. Kori has never experienced a microsleep when driving. He avoids daytime naps and he does not regularly consume caffeine. There are no features to suggest narcolepsy.
Around 40 million (Sleeping Disorder Statistics, 2012) Americans suffer from chronic sleeping disorders, regardless of their age. Some common disorders are insomnia, narcolepsy, and sleep apnea. Individuals who suffer from insomnia have a difficult time falling asleep and staying asleep, resulting...
On week days I tend to be in bed around 11:00pm and plan to be asleep by midnight. Falling asleep by midnight is usually accomplished on each weeknight, with an occasional late night study session. I am fortunate enough to be able to sleep later than I have been in past semesters. We have not had our usual 6:00am morning team lifting for crew and I do not have many early classes. It is not difficult for me to wake up once I hear my alarm in the morning. When I know I have something to complete or somewhere to be I am able to jump right out of bed and get ready. While filling out my Stanford Sleepiness rating times, I was able to give myself scores bet...
Kales, A. (1972). The evaluation and treatment of sleep disorders : Pharmacological and psychological studies. In M. Chase (ed.)The Sleeping Brain. Los Angeles : Brain Information Service.
National Heart, Lung, and Blood Institute. (2012, July 10). Retrieved April 23, 2014, from National Heart, Lung, and Blood Institute: http://www.nhlbi.nih.gov/health/health-topics/topics/sleepapnea/
The sleep study was limited somewhat by a reduced total sleep time seen and of note there was no supine sleep sampled. In the non-supine sleep seen, there was a moderate degree of obstructive sleep apnoea with an AHI of 16 events/hr which has significantly worse in REM sleep.
The patient is currently here for a CPAP titration study following a home cardiorespiratory monitoring test that revealed symptoms consistent with OSA. CPAP titration protocol was explained upon arrival. The patient expressed prior to hookup that he was unsure if he would be able to tolerate CPAP therapy due to nasal congestion that also affects his ability to initiate/ maintain sleep.
Ken was seen today now 18 months into therapy for his moderate degree obstructive sleep apnoea. He is doing very well and has persisted with his nasal mask and he is no longer noticing any mouth breathing. He is using his machine seven hours a night and his residual AHI of 0.8 events/hr is suggesting complete control of his obstructive sleep apnoea. There is a high mask leak indice which he believes is more so from his mask than mouth and this seems to have resolved with tightening of the straps.
Sleep and dreams have defined eras, cultures, and individuals. Sigmund Freud’s interpretation of dreams revolutionized twentieth-century thought. Historical archives record famous short sleepers and notable insomniacs—some accounts reliable, some not. When Benjamin Franklin counseled, “Early to bed, early to rise, makes a man healthy, wealthy, and wise,” he was using sleep habits to symbolize his pragmatism.
Obstructive sleep apnea (OSA) can be defined as a transient cessation in breathing (apnea) or a reduction in breathing amplitude (hypopnea) as a result of collapsed or obstructed upper airway; both can cause notable arterial hypoxemia and hypercapnia. The apnea/hypoapnea index (AHI) categorizes OSA patients into three groups: mild (5-15), moderate (15-30) and severe (>30) whereas the number indicates the total number of apnea/hypoapnea episodes per hour of sleep {Badran et al., 2014, #73409}. Although many sleep clinics adopted this diagnostic tool, diagnostic variability remains {Caples et al., 2005, #48595}. AHI measures the frequency of disordered breathing events but does not quantify other processes involved in the pathophysiology of OSA, such as the degree of oxygen desaturation. For instance, the increasing length of apnea/hypoapnea events will likely increase oxygen desaturation events which is very stressful and have more severe pathological impact than shorter ones. Paradoxically, lengthening of apnea/hypoapnea events can lead to a decrease in AHI {Kulkas et al., 2013, #24356}.
Have you ever had the fear of not being able to breathe while sleeping? Well, many Americans have to deal with this every single night. Many people are undiagnosed and unware that they even have this condition. If untreated, sleep apnea can cause people who have it to do poorly in everyday activities (“Sleep Apnea”). Today, I am going to be informing you about sleep apnea. I chose to research sleep apnea because I had never heard of it before and I didn’t know it was as common as it is. I also love researching new topics and discovering things that I don’t know much about. I’ve prepared for this speech by researching my topic and using many websites and sources for finding information about sleep apnea. First, I will be talking about what sleep apnea is, the two types of sleep apnea there are, and the symptoms and effects of it as well. Second, I will be talking about what causes sleep apnea, and the treatments for it. Finally, I will be telling you about
The participants were prescreened for past usage of taking medication that altered their sleep, any past/current diagnosis of a sleep disorder, and any mental disorders. Twenty-seven adults were considered ineligible after taking the screening test. The participants were also excluded if they didn’t return for their follow up session, if the excessively napped, and if there was an equipment failure leading to no sleep
During the multiple sleep latancy test performed the following day, no sleep was recorded indicating