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Research paper on narcolepsy
Research paper on narcolepsy
Narcolepsy research paper
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Thank you for referring Kori Stankovic, a 24 year-old software installer who is non-smoker, consumes alcohol on average a couple of standard drinks every couple of days and does not engage in any recreational drug use. Kori does not take any regular medication. Unfortunately the referral letter was not available at the time of review however I gather the reason for referral is that Kori has been suffering with sleep onset insomnia and unrefreshing sleep. He usually retires to bed at 10.30pm but will generally not be able to sleep for at least an hour. On occasions he will then listen to some soft music and then make another attempt to fall asleep. Often he does not fall asleep until 1.30am to 2.00am. There is no history or snoring, witness apnoeas or nocturnal choking episodes but Kori's sleep is fragmented but there is no sleep maintenance insomnia. He reports general restless but no specific restless legs symptoms as such. When he awakes at 7.00am, he consistently feels unfreshed and he is aware of some daytime somnolence although the Epworth Sleepiness score is normal at 7/24. Kori has never experienced a microsleep when driving. He avoids daytime naps and he does not regularly consume caffeine. There are no features to suggest narcolepsy. …show more content…
LFTs reveal an elevated ALT of 102, AST 838 and gamma GT 56. Viral hepatitis serology is negative. Fasting glucose and cholesterol are slightly
Thank you for referring Ransley Mascurine, a 55 year-old gentleman who currently works as a counsellor and is also an avid body builder. Ransley is a non-smoker and of late, has been consuming alcohol two to three standard drinks each evening. The only regular medication is Lipitor.
A 46-year-old Hispanic man presented to the clinic for a routine physical and a review of his recent laboratory studies. His medical history was significant for high cholesterol. His daily medication regimen included a multivitamin, aspirin 81 mg, and simvastatin (Zocor) 40 mg for high cholesterol. His family history was remarkable for type 2 diabetes, hypertension, and hyperlipidemia in his mother, maternal grandmother, maternal aunt, and maternal uncle. Physical examination revealed a height of 69 inches, weight of 199 pounds, body mass index (BMI) of 29.4 kg/m2, waist circumference of 36 inches and blood pressure of 125/72 mm Hg. Initial laboratory results were: total cholesterol 195 mg/dL; triglycerides 136 mg/dL; low-density lipoprotein cholesterol (LDL), 124 mg/dL; high-density lipoprotein cholesterol (HDL), 44 mg/dL; and fasting plasma glucose (FPG), 119 mg/dL. Complete blood count, renal function and liver function tests were all normal. Additional studies included an oral glucose tolerance test (OGTT) of 157 mg/dL and hemoglobin A1c (HbA1c) of 5.9%. The patient’s physical exam was unremarkable with no abnormalities noted.
This case study is about Abdul Chidiac, a 51 year old male, married with 4 children. He had a medical history of hypertension, hypercholesterolaemia and cirrhosis with two admissions in the last six months. He is a smoker and drinks beer, 5-6 bottles per day. As Carithers & McClain (2010) explained the patient’s medical history is another indicator of the risk for cirrhosis; the progression to cirrhosis is adaptable and may take time over weeks or many years. Cirrhosis is a liver disease characterized by permanent scarring of the liver that interferes with its normal functions including alcoholism. Most people who drink large amounts of alcohol cause harm to the liver in some way (Heidelbaugh & Bruderly, 2006). The cause of cirrhosis is not yet known, but the connection between cirrhosis and excessive alcohol ingestion is established (Jenkins & Johnson, 2010). Common causes of cirrhosis include: alcohol abuse, hepatitis B infection, hepatitis C infection and non-alcoholic fatty liver disease (NAFLD) and non-alcoholic steatohepatitis (Schuppan & Afdhal, 2008).
Korsakoff’s syndrome is a brain disorder that is related to heavy alcohol use over a long period of time. This disorder is caused by a lack of Thiamine, or vitamin B1. Excessive amounts of alcohol use lead to Thiamine deficiency, which affects the brain and nervous system. Thiamine deficiency can be caused by poor eating habits, as heavy drinkers typically do not have nutritional diets that fulfill their vitamin needs. Alcohol can also disrupt the process in which Thiamine is changed into the active form, Thiamine Pyrophosphate. Alcohol also inflames the stomach lining, causing vomiting; again, this affects the body’s absorption of key vitamins. The effect alcohol has on the liver also affects the storage of these vitamins. Korsakoff’s syndrome is also related to another brain disorder, Wernicke-Korsakoff syndrome. This syndrome involves the Korsakoff syndrome and also Wernicke. Wernicke’s syndrome involves undernutrition, jerky eye movements, poor balance, and memory loss, which is caused by heavy alcohol consumption. If this condition is...
THESIS STATEMENT: Narcolepsy is a sleep disorder that has a specific medical definition, life-changing symptoms, and there are ways in how people treat it.
J.P., a 58 year old female, presents to the Emergency Room on March 18th. She has a past medical history of cervical cancer, atheroembolism of the left lower extremity, fistula of the vagina, peripheral vascular disease, neuropathy, glaucoma, GERD, depression, hypertension, chronic kidney disease, and sickle cell anemia. She complains of right lower extremity pain accompanied by fatigue, a decreased appetite, increased work of breathing, burning urination, and decreased urine output for three days. Upon admission, a complete physical assessment was performed along with a blood and metabolic panel. The assessment revealed many positive and negative findings.
Chronic sleep loss is becoming more common in modern culture and less restricted to sleep-deprived diseases such as insomnia. Suggested to be the result of a number car, industrial, medical, and other occupational accidents, sleep deprivation is beginning to be recognized as a public concern. As a result, the Centers for Disease Control
Saitz, Richard, M.D. "Unhealthy Alcohol Use." New England Journal of Medicine (2005): 596. Web. 7 Apr. 2014.
Millions of people suffer from the same tossing and turning every which way, getting their sheets all disarranged and their minds abundantly worse. Patients often report indications of insomnia while sitting in the family health clinic. Insomnia traits include hindrance, falling asleep, continuing to awaken, and rejuvenating before wanted. One may suffer from insomnia if one shows signs of an increased difficulty in attentiveness, decreased communal or scholastic skills, and a diminished mood or enthusiasm. Foldvary-Schaefer 111.
Pulmonary complications of smoked substance abuse. West Med 152: 524-530.
Narcolepsy has been above looked for years beforehand knowing a patient has the illness, it is a quickly producing awareness and is continually altering people and their families lives. With nap materializing to be not merely the ultimate pastime, but additionally a survival imperative, the earth of nap scutiny is quite large, bragging countless disparate spans of study. By scrutinizing phenomena like nap disorders neurobiologists can yearn to comprehend the mechanisms of normative nap, in supplement to perfecting treatment for suffers. Narcolepsy is one such disorder that affects an approximated 250, 000 or 1 in 2000 Americans; comparable numbers are approximated for Parkinson's or countless sclerosis (mayo-foundation). An comprehensive, nevertheless oftentimes misdiagnosed illness (fewer than 50, 000 are cognizant of their condition), narcolepsy can be delineated by chronic daytime sleepiness, cataplexy, nap paralysis, and hypanogic hallucinations (rare-disease). The last three of the tetrad of symptoms additionally transpire in non-narcoleptic individuals; nap episodes are the main determinant in diagnosis. Merely 20 to 25 percent of narcoleptics tolerate from all four symptoms (mayo-foundation). This paper has countless goals, all of that involve elucidating the illness and its symptoms in disparate contexts. In order to do this nap will main be elucidated in a slight detail, pursued by a biological and psychological treatment of narcolepsy. Scutiny of narcolepsy and its implications for the upcoming displays steps to be grabbed in order to garner a larger understanding this particular brain/behavior relationship.
Wilson, J.F. (2005). Is sleep the new vital sign? Annals of Internal Medicine, 142 (10), 877-880.
Kales, A. (1972). The evaluation and treatment of sleep disorders : Pharmacological and psychological studies. In M. Chase (ed.)The Sleeping Brain. Los Angeles : Brain Information Service.
Polysomnography (PSG) will be used to evaluate sleep disturbance. To ensure eligibility, the study coordinator will go through a screening checklist. If at least one criterion is not met, the patient will be excluded from participating. If the participant meets eligibility criteria, they will be invited to participate and the phases of the study will be explained. Upon informed consent, demographic data will be collected from the patient or from the patient chart. Additionally, a fecal calprotectin kit will be administered to each participant to confirm remission status. To prepare for PSG, participants will be instructed to keep a 2-week sleep diary prior to overnight evaluation. Finally, participants will complete 3 questionnaires before their sleep appointment and submit them, along with the sleep diary: (1) Functional Assessment of Chronic Illness Therapy-Fatigue questionnaire; (2) Beck Depression Inventory-II; and (3) Pittsburgh Sleep Quality Index questionnaire. PSG will take place in a level-one sleep laboratory at the Edmonton General Continuing Care Centre. Following PSG observations, the data will be analyzed via statistical software to determine if the fatigue reported by patients with IBD is related to sleep
Sleep and Sleep Disorders. Centers for Disease Control and Prevention, 1 July 2013. Web. 7 May 2014. .