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Heart anatomy and physiology
Anatomy of the Heart and Cardiovascular System
Atrial septal defect
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An atrial septal defect is a congenital birth defect of the heart in which there is a hole in the septum that divides the atria. The hole can vary in size and may close on its own or may require surgery. The Centers for Disease Control and Prevention recently estimated that each year about 1,966 babies in the United States are born with an atrial septal defect. About 5% to 10% of all coronary heart disease are atrial septal defects and are responsible for about 30% of the congenital heart defects diagnosed in adults. Atrial septal defects are twice as common in females as in males. Most atrial septal defects occur periodically as a result of spontaneous genetic mutations even though hereditary forms have been found, also linked extra-cardiac congenital defects are present in 25% of infants with atrial septal defect, and about one third have a hereditary syndrome. …show more content…
In many cases, it may not be diagnosed until adulthood. What happens in smaller atrial septal defects is that minor blood shunting within the heart occurs and it has no hemodynamic significances. Larger defects are usually accompanied with significant shunting, which can lead to volume overload of the right atrium, right ventricle, and pulmonary arteries. The degree of left-to-right shunting depends on the size of the atrial septal defect, the relative compliance of both ventricles, and the pulmonary and systemic vascular resistance. If left untreated, this can cause pulmonary hypertension, right ventricular failure, reduced right ventricular compliance, and has the potential to cause right-to-left shunting.
Three nursing diagnoses for atrial septal defects are as follows:
1. Activity intolerance related to fatigue, generalized weakness and lack of adequate oxygenation.
2. Decreased cardiac output related to cardiac dysfunction.
3. Risk for disorganized infant behavior, risk factor is invasive
When MVP occurs, the left ventricle contracts, one or both flaps of the mitral valve flop or bulge back (prolapse) in the left atrium, this prevents the valve from forming a tight seal. As a result, blood may leak back into the atrium which is referred to as regurgitation (nhlbi.nih.gov).
“Hypoplastic left heart syndrome accounts for 9% of all critically ill newborns with congenital cardiac disease, causing the largest number of cardiac deaths in the first year of life.(2) ” HLHS is a severe heart defect that is present at birth. HLHS combines different defects that result in an underdeveloped left side of the heart. This syndrome is one of the most challenging and difficult to manage of all of the congenital heart defects. Multiple portions on the left side of the heart are affected including the left ventricle, the mitral and aortic valve, and the ascending aorta. These structures are greatly reduced in size, or completely nonexistent causing the functionality of the left heart to be reduced, or non-functional all together.
Cardiomyopathy, by definition, means the weakening of the heart muscle. The heart is operated by a striated muscle that relies on the autonomic nervous system to function. Cardiomyopathy is diagnosed in four different ways based on what caused the illness and exactly what part of the heart is weakened. The four main types of cardiomyopathy are dilated cardiomyopathy, hypertrophic cardiomyopathy, restrictive cardiomyopathy, and arrhythmogenic right ventricular dysplasia. One other category of cardiomyopathy that is diagnosed is “unclassified cardiomyopathy.” Unclassified cardiomyopathy is the weakening of the heart that does not fit into the main four categories.
This syndrome increase blood flow causes the heart to pump blood to the lungs at an increasing rate and destroys the blood vessels in the lungs. Several Heart defects that causes disorder is ventricular septal defect (VSD), atrial septal defect (ASD), Patent ductus arteriosus (PDS), and Atrioventricular canal defect (ACD) (Mayo Clinic,2016) This hole usually causes symptoms that include blue or gray skin pigments, shortness of breath, extreme fatigue, chest pains, racing or skipped heart beats, and dizziness. Other symptoms include coughing up blood, swelling in the abdominal region, and numb and/or enlarged fingers and toes. Some of the way ER syndrome can be diagnosed are Chest X-ray are used for heart and pulmonary artery enlargement. Electrocardiogram (ECG) electrical activity of the heart that help test for heart defect that are caused by ES, Echocardiogram is normally used for listing to sound of the heart during, but during ER testing it helps to see if the patient have a heart defect, Magnetic resonance imaging (MRI) is used to take images of blood vessels and lungs and blood test is use to check blood count, which ES would make it
The Tell Tale Heart and Greasy Lake have interesting characters to analyze. Edgar Allen Poe’s Tell Tale Heart has an eerie and dark tone that Poe’s literary work is known for. Greasy Lake by T.C. Boyle starts out with hardcore yet naïve teenagers looking to had a good time. However, their naivety and immaturity will led them into a very bad situation.
The commonest defect is a narrowing of the main artery from the heart aortic coarctation. A regular ultrasound examination of the heart ... ... middle of paper ... ... s are relatively limited. So far, researchers think it will be possible to increase the final height by 5-10 centimeters, depending on the duration of treatment.
... as the heart, major blood vessels, and airways) toward the other side of the chest. The shift can cause the other lung to become compressed, and can affect the flow of blood returning to the heart. This situation can lead to low blood pressure, shock, and death.
Tell Tale Heart is a short horror story by E.A. Poe that is told from the first person perspective and describes the murder of an old man. The main character plots the crime because he (supposing the narrator is male) is irritated by the old man’s “evil eye”. The narrator kills the old man in his sleep, dismembers the body and hides the corpse parts under the floorboards. The main character is not suspected until he confesses the murder to the police believing everyone can hear the beating of the dead man’s heart from under the floor. Tell-Tale Heart is not a confession but an apology. The murderer tries to prove that the hideous crime, no mater how irrational it might seem to the readers, was planned and carried out in the calculated and premeditated manner. The narrator tries to convince the readers that he was conscious of his motives, actions, and intentions. What is more, he stresses that there was no trace of permanent or temporary mental disorder, let alone insanity. However, the choice of the point of view, tone and mood of the Tell-Tale Heart allow Poe to create the opposite effect and convince the readers that the story is an account of a madman. The psychological effect of the first-person narrative, the tone and symbolism let Poe enhance the gruesome effect of the story. The point of view chosen by Poe also makes readers feel as if the insane narrator addresses every reader personally. A vide range of stylistic devices is employed to make the story frightening from the very beginning.
Hypertrophic cardiomyopathy is an inherited disease that affects the cardiac muscle of the heart, causing the walls of the heart to thicken and become stiff. [1] On a cellular level, the sarcomere increase in size. As a result, the cardiac muscles become abnormally thick, making it difficult for the cells to contract and the heart to pump. A genetic mutation causes the myocytes to form chaotic intersecting bundles. A pathognomonic abnormality called myocardial fiber disarray. [2,12] How the hypertrophy is distributed throughout the heart is varied. Though, in most cases, the left ventricle is always affected. [3] The heart muscle can thicken in four different patterns. The most common being asymmetrical septal hypertrophy without obstruction. Here the intraventricular septum becomes thick, but the mitral valve is not affected. Asymmetrical septal hypertrophy with obstruction causes the mitral valve to touch the septal wall during contraction. (Left ventricle outflow tract obstruction.) The obstruction of the mitral valve allows for blood to slowly flow from the left ventricle back into the left atrium (Mitral regurgitation). Symmetrical hypertrophy is the thickening of the entire left ven...
This condition can be treated various ways depending on “the type and frequency of arrhythmias, associated symptoms…, and the presence of structural heart disease” (Cleveland Clinic, 2014). Some patients may not need treatment at all, since they show not symptoms, since sometimes this condition can be naturally eliminated over the first year of life, but may still be required to have regular schedule appointments with the physician so the patients can be monitored. If symptoms are prevalent, the different treatment methods include a pacemaker, defibrillator, surgery, and medicine.
Most often the disease starts in the left ventricle, and then often spreads to both the atrium and right ventricle as well. Usually there will also be mitral and tricuspid regurgitation, due to the dilation of the annuli. This regurgitation will continue to make problems worse by adding excessive volume and pressure to the atria, which is what then causes them to dilate. Once the atria become dilated it often leads to atrial fibrillation. As the volume load increases the ventricles become more dilated and over time the myocytes become weakened and cannot contract as they should. As you might have guessed with the progressive myocyte degeneration, there is a reduction in cardiac output which then may present as signs of heart failure (Lily).
The aim of this essay is to critically analyse a clinical incident involving an adult with Congenital Heart Disease (CHD). I will define reflection, then select a reflective model and critically reflect on the incident demonstrating my new found knowledge. Lastly, I will suggest how nursing practice should change to improve the care of this group of patients.
Oxygen was first admitted to the client with chest pain over 100 years ago (Metcalfe, 2011). Chest pain is a large bracket that can contain many different conditions, but for the purpose of this analysis it is focused manly upon a myocardial infarction. A myocardial infarction is mainly referred to as a heart attack, and occurs when one or more coronary arteries leading to the heart reduce or completely stop blood flow (Tuipulotu, 2013 ). Administering high concentrations of oxygen to patients with chest pain is now embedded in guidelines, protocols and care pathways, even with a lack of clear supporting evidence (Nicholson, 2004 ). High concentration of oxygen means that up to 60% is administered (Knott, 2012). More recent research has suggested that the use of oxygen in this scenario is unnecessary and can lead to unwanted side effects, especially in normoxic cardiac patients (Moradkham & Sinoway, 2010 ). The aim of this comparative analysis is to dismantle and understand both the benefits and risks of the commonly known practice of administration of oxygen to the client with chest pain. Through completing this analysis using recent and appropriate evidence a more improved practice can be given and understood.
-Congenital defect in which the lateral halves of the palate fail to fuse during embryonic development. It may be localized to the uvula, the soft and/or hard palate, or the lip. Combined cleft lip and palate is more common in males, whereas isolated cleft palate occurs more frequently in girls. Corrective surgery is usually successful if approached after 18 months of age.