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Disabilities in modern society
Disabilities in modern society
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command in the rise of muscle tone is sent to the spinal cord from the nerves from the muscle itself, which are called the sensory nerve fibers. The muscles of the patient appear stiff and rather than create smooth and swift movement, appear rigid and jerky. When contracting or moving a muscle to create movement, some muscles are turned on, while others are turned off, making it extremely challenging to move normally. Spasticity, depending on severity, can greatly affects one’s ability to walk, stand, sit in the correct posture and even perform daily tasks such as bathe, get dressed, eat, write, and using the restroom. It can also have an impact on speech, causing the patient to have slow oral movements, slurred speech, and a tight or hoarse …show more content…
voice. A personal account of spastic cerebral palsy said, “I have always liked writing. Having high-tone CP, many may pre-judge me on my body movements. My writing [by comparison] can take 10 minutes or 10 hours to construct and I am judged on the final outcome — not how I got there...They see my body constantly moving, they see me in the wheelchair, etc. Automatically the mind goes into preconceived judgment on my intellectual ability…” (2014 Dan Reimold) Within the category of spastic CP, there is an even more distinct category called, spastic quadriplegia. This very specific group affects the entire body. Patients suffering from this are unable to walk and have moderate to severe intellectual disabilities. The next type of CP is Athetoid Palsy, which affects 10 to 20% of patients.
It is associated with the damage of the basal ganglia, which is essential to coordination of movement. and usually develops around 18 months of age. The symptoms of Athetoid CP are low, involuntary movement, such a thrashing or writhing of the hands, arms, and legs. Involuntary grimacing of the face, drooling, and inability to hold or perform deliberate actions may also occur. In some cases, speaking, eating, and swallowing can also be impaired, resulting in lack of nutrients. Regarding muscles, this disorder can cause too loose or too tight muscle tone, leading to possible muscle spasms. Patients can also develop complications in cervical spine, hip, and feet, usually resulting them to be entirely disabled. Fortunately, a patient’s mental capacity is usually not altered in effect to this specific type, in which most patients have normal or potentially above average of intelligence. Symptoms such as fetal stroke causing infant’s brain to bleed, blood clots in the placenta causing to block the infant’s circulation, or severe Jaundice can all result in athetoid Cerebral Palsy. This is a personal account of a person affected by athetoid Cerebral Palsy, “As a teen with CP it was easy for me to get overlooked, discouraged, or even patronized simply because I wasn't considered "normal." This became especially apparent when doctors began prescribing "special devices" to help me re-establish or maintain a …show more content…
"suitable range of motion." These devices were to be worn while doing everyday activities, including school-time activities...Because my CP mostly affects my legs and the way I walk, I had to wear knee-high, white-plastic, custom-made leg braces at all times during my first couple of years of high school.” (2015 Children’s Healthcare of Atlanta Inc.) Ataxic Palsy is the rarest type of CP, therefore, only affects about 5% of patients diagnosed with Cerebral Palsy. This specific type involves a loss of muscle coordination such as depth perception and balance. Patients suffering from Ataxic Palsy often walk with long unusual strides due to impaired depth perception and struggle doing activities such as inserting a coin into a slot, which involves precision movements. Intention tremor is a symptom that is common within patients, when reaching for an object, the fingers, hand, or arm begins to quiver or shake; worsening the closer they are to the object. Mental retardation can be a factor of this type of Cerebral Palsy but intelligence as a whole is usually unaffected. Similar to the other types of CP, Ataxic can be caused by abnormality of the brain, usually formed from infections of the mother or the unborn baby or by lesions, holes, in the brain’s white matter that is essential to signaling the nervous system. The last type of Cerebral Palsy is Mixed CP, which is when symptoms of spasticity and athetoid occur together. Signs of spasticity will usually be detected before because involuntary movement of athetoid usually doesn’t occur until nine months to three years. When symptoms of both types of Cerebral Palsy collide, muscles of patient may appear spastic as well as limp. Having both characteristics, make it extremely difficult to create treatments for mixed CP. Mental retardation, speaking impairment, Strabismus, known as “cross-eyes or “lazy eye”, and seizures caused by unusual electrical activity in the cerebrum are very common factors of mixed CP. Cortical blindness is another symptom that occurs in patients affected by mixed CP, in which the eyes appear unaffected by the actual vision is damaged. A cause of this specific type is a condition called Rh incompatibility, which the mothers’ and fetus’ blood type is opposite, resulting in the mother taking in anti-bodies against the baby’s blood. Maternal infections, inflammatory pelvic diseases, and thyroid problems within the mother can all lead to mixed Cerebral Palsy. One of the most complicating factors of Cerebral Palsy is mental impairment.
Patients affected by this can have a difficult time comprehending and pronouncing words, staying focused on one task, limiting being able to multi-task, such as read and speak simultaneously, and inability to retain information as quickly. Though these characteristics of intellectual disability may occur, the patient is still able to improve and learn new skills, just at a slower pace. Diagnosis for this is through a process of observation between kids their age. Things such as being able to feed, bathe, and clothe one self, communicate with other children, and ability to solve, understand, and react to daily life
problems. Seizures are symptoms that affect almost half of the patients affected with Cerebral Palsy. A specific type, tonic-clonic seizures, which can cause the patient to go unconscious, twitch and tighten muscles of the body, cry or groan, and jerk arms, legs, and head. Partial seizures, also occur, in which contractions can appear on one side of the body, skin begins to feel numb or tingly sensation, unusual head or eye movement, abdominal pains, nausea, and a sense of mood change. These can be caused by unmonitored bursts of electricity that interfere or disrupt the brain’s normal functions or routine of electrical activity. There are many factors that contribute to neurologic disorder of Cerebral Palsy. Though, each patient’s symptoms may appear different, reasoning for the symptoms may appear different as well. Ultimately, CP is caused from damage in the brain or brain malformation that occurs before, during, or after brain development. Depending on the severity of the damage to the brain along with the location, a vary of Cerebral Palsy types can develop. Ranging from Spastic Cerebral Palsy to Mixed Cerebral Palsy. Factors that can cause CP is inadequate oxygen reaching baby during pregnancy and birth, maternal infections within the mother during pregnancy, and premature birth. Diseases within the mother that can lead to Cerebral Palsy are: • German Measles: a viral disease that cause major birth defects
In the beginning phases of muscle contraction, a “cocked” motor neuron in the spinal cord is activated to form a neuromuscular junction with each muscle fiber when it begins branching out to each cell. An action potential is passed down the nerve, releasing calcium, which simultaneously stimulates the release of acetylcholine onto the sarcolemma. As long as calcium and ATP are present, the contraction will continue. Acetylcholine then initiates the resting potential’s change under the motor end plate, stimulates the action potential, and passes along both directions on the surface of the muscle fiber. Sodium ions rush into the cell through the open channels to depolarize the sarcolemma. The depolarization spreads. The potassium channels open while the sodium channels close off, which repolarizes the entire cell. The action potential is dispersed throughout the cell through the transverse tubule, causing the sarcoplasmic reticulum to release
Flaccid Dysarthria falls within one of two major categories of motor speech disorders. Motor speech disorders are classified as either dysarthria’s or apraxia’s. Flaccid Dysarthria, which has to do with damage to the lower motor neurons, is only one of six categorized dysarthria’s, the others being as follows: Spastic Dysarthria, Ataxic Dysarthria, Hyperkinetic Dysarthria, Hypokinetic Dysarthria, and Mixed Dysarthria. Features of each of these dysarthria’s are distinctive due to the site of damage and can help with the diagnosis of the particular dysarthria.
Flaccid dysarthria results from damage to the lower motor neurons (LMN) or the peripheral nervous system (Hageman, 1997). The characteristics of flaccid dysarthria generally reflect damage to cranial nerves with motor speech functions (e.g., cranial nerves IX, X, XI and XII) (Seikel, King & Drumright, 2010). Lower motor neurons connect the central nervous system to the muscle fibers; from the brainstem to the cranial nerves with motor function, or from the anterior horns of grey matter to the spinal nerves (Murdoch, 1998). If there are lesions to spinal nerves and the cranial nerves with motor speech functions, it is indicative of a lower motor neuron lesion and flaccid dysarthria. Damage to lower motor neurons that supply the speech muscles is also known as bulbar palsy (Pena-Brooks & Hedge, 2007). Potential etiologies of flaccid dysarthria include spinal cord injury, cerebrovascular accidents, tumors or traumatic brain injury (Pena-Brooks & Hedge, 2007). Possible congenital etiologies of flaccid dysarthria include Moebius syndrome and cerebral palsy. Flaccid dysarthria can also arise from infections such as polio, herpes zoster, and secondary infections to AIDS (Pena-Brooks & Hedge, 2007). Additionally, demyelinating diseases such as Guilian-Barre syndrome and myotonic muscular dystrophy can also lead to flaccid dysarthria (Pena-Brookes & Hedge, 2007). The lower motor neuron lesion results in loss of voluntary muscle control, and an inability to maintain muscle tone. Fasciculations, or twitching movements, may occur if the cell body is involved in the lesion (Seikel et. al., 2010). The primary speech characteristics of flaccid dysarthria include imprecise consonant production, hypernasal resonance, breathiness, and harsh voice (...
ASC they may need more reassurance and physical contact and different strategies to aid their communication, such as sensory breaks, to help their focus as they may have a short attention
Many people have heard the term cerebral palsy and may have a personal perception about the appearance and effects of this
Perception is an intangible part of every being. It cannot be explained, defined, or nailed down the way that most scientists would like. In some ways, perception can be taught-a person's circumstance and background would cause him or her to perceive a situation in a particular way. In other ways, perception is unpredictable and ever changing. Even here, attempting to describe the indescribable, there are flaws in the last two sentences because they are based on the writer's perceptions of perception. It is too subjective for a "scientific" definition. What does it mean for a person suffering from bradykinesia? If the individual understands the condition, she will realize that the perceptions she has are not always correct. She may perceive herself to be making a fist, or spreading her fingers, but in fact she may not have accomplished this. (1) A blind and deaf person may have perceptions about the world around her. Most likely, her only correct perceptions are those perceptions about herself such as: "I am moving my arm," or "I am swinging my legs." The external stimuli are ineffective in this person, whereas a person with bradykinesia can only react completely and at a normal speed to external stimuli. Because of damage to signal pathways, the internal stimuli are ineffectively activated. (1)
Cerebral Palsy (CP) is a condition marked by impaired muscle coordination and other disabilities, which causes damage to the brain before and during birth. Cerebral palsy is a static disorder of the brain, not a progressive disorder. This mean that the disorder or disease process will not get worse as time goes on. Nor are the motor disorders associated with cerebral palsy temporary. (Miller and Bachrach pg. 3) Cerebral Palsy affects the nervous system by having dysfunctions, in movements such as, learning, hearing, seeing, and thinking. During the first 3 to 5 years of a child's life Cerebral Palsy occur because the baby's brain is still developing. (CP is one of the most common congenital (existing before birth or at birth) disorders of childhood). Spastic, athetoid, ataxic and dystonic are all different types of Cerebral Palsy. Majority of circumstances with children having CP are unknown, then again numerous results show problems during pregnancy in which the brain is damaged or doesn't develop normally. “This can be due to infections, maternal health problems, a genetic disorder, or something else that interferes with normal brain development.” Cerebral palsy is also caused by injuries and abnormalities of the brain; as the baby grows in the womb these problems occur. Some causes may lead to problems with brain development which include:
Patients whose lesion is in the Cerebrum & Cerebellum will experience loss of balance and coordination, difficulty speaking, and frequent limb trembles. Speech difficulties vary from slurred words, long pause between words, and swallowing problems. Patients whose lesion is in the Motor nerve tracts will experience weakened and stiffened muscles, blurred vision or vision impaired, and urinary problems. Weakened and stiffened muscles causes walking disabilities and painful feeling of muscle spasms in 6 to 10 people. Patients whose lesion is in the Sensory nerve tract will experience sensory alternations, fatigue, cognitive and emotional dysfunction, and loss of sexual interest. Difference sensations experienced are numbness, itching, burning, stabbing, or tearing pains.
Some characteristics of DS are: deep folds at the corners of the eyes, hypotonia, short stature, flexible joints, small oral cavity and heart defects (Taylor, Richards, & Brady, 2005). Most individuals with DS have a moderate intellectual disability, although there is a range of disability, from severe to high functioning (IQ above 70). Since DS is a birth defect and not a disease, there are no treatment options. Improvement can be made through physicians, special education, physical therapy, speech therapy, occupational therapy, and psychol...
Clinical diagnosis of PD is currently solely dependent on the presentation of the symptoms by the patient which reflect a deficiency of striatal dopamine caused by the destruction of the cells in the substantia nigra. Imaging and other laboratory techniques can be used to rule out other disorders, but are not necessary for the actual diagnosis of PD. The first sign of PA is usually bradykinesia. Movements are usually quite slow. Routine activities may require deliberate planning and thought for execution. Difficulty initiating movements or akinesia, may also be present. Rigidity in the flexors is also present. This is due to an exaggerated response to normal proprioceptive return from the somatic musculature. A resting tremor of 3-6 Hz is also a prominent feature of PD. This may cause difficulties in handwriting as a symptom. Impaired postural reflexes is also a presenting feature in PD. Patients can easily lose their balance when pushed slightly, and may need to be caught to keep from falling. These signs can be tested by observing the patients walking, getting out of deep chairs, and performing rapid repetitive movements. Increased disturbances in cognitive abilities can also show evidence of PD. Even with all these signs of PD, it may be present and undiagnosed f...
The National Institute of Neurologic Disorders defines learning disorders as a; condition that either prevents or significantly hinders somebody from learning basic skills or information at the same rate as most people of the same age. It is important to note that this does not mean that the individual has less intelligence; it means that their brain or body is wired in a way that hinders or prevents development of a certain tasks. Many people often confuse learning disabilities with mental retardation but while both words describe similar conditions, the term learning disability and learning disorder are used to describe conditions that affect development in certain cognitive areas in otherwise healthy developed individuals. Millions of people are affected in differing severity throughout the world with specific neurologic based learning impairments.
“Before God we are all equally wise - and equally foolish.” (Einstein). Developmental disability, better known as intellectual disability, or mental retardation is a disorder that causes individuals to preform at below average levels (“Intellectual”). This disorder is characterized by continued infant-like behavior, decreased learning ability, failure to meet the markers of intellectual development, inability to meet educational demands, and a lack of curiosity (“Intellectual”). Some people with intellectual disabilities are able to live normal lives, while others may require assistance. Most people with intellectual disabilities have the same capacity to preform the same task as those without intellectual disabilities.
Intellectual disability (ID) and related terminology have evolved over time to reflect the legal and social gains made by individuals with such a disability and their families. ID is characterized by significant limitations in intellectual functioning (e.g., reasoning, learning, and problem solving); significant limitations in adaptive behavior (i.e., conceptual, social, and practical skills in everyday life); and onset in childhood (before the age of 18 years; American Association on Intellectual and Developmental Disabilities (Disabilities, 2017). Intellectual disability is a particular state of functioning that begins in childhood and is characterized by limitations in both intelligence and adaptive skills (Janet W. Lerner, 2015).
This can include things that they hear on a normal basis, like commercials or songs. Uneven language development is one of the biggest red flags that indicate ASD. For example, a normal child consistently learns new words and continues to learn how to arrange them into cohesive statements, whereas a child with ASD learns a few words and then has long periods in between new words learned.... ... middle of paper ... ... Early signs of Autism spectrum disorder.
During development, a child develops in a couple differnet ways. Physically, emotionally, and cognitively are the three that come to mind. When a child has developmental delays, there are many things that can be happening preventing the child from developing. Sometimes muscles aren't moved as often as they should be and become immobile. When a child picks up an instrument for the first time, he is not naturally coordinated to play that instrument. Children with that lack in motor skills can exercise, especially their hands, without even knowing it. There are...