Hyperventilation Lab Report

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. In the activity when Hyperventilation occurs there is not enough carbon dioxide in the blood. During hyperventilation the tidal volume is larger, which removes the carbon dioxide faster than it can replenish it. This is also called respiratory alkalosis; the renal system is able to compensate for respiratory alkalosis by increasing the partial pressure of the carbon dioxide levels and then decreasing the pH levels. In the experiment when we went from normal breathing to hyperventilation our Min Pco2 went from 40 to 25.94. Our pH then went from 7.40 to 7.58 max pH. When the hyperventilation stopped and the trace flat lined, this showed that the breathing was suspended. The body had to restore the Pco2 levels back to the normal value. During this activity we had the normal breathing at 40 Pco2 max and 7.39max pH and then we went to rebreathing and it jumped to 52.09 max Pco2 and 7.42 max pH. The rebreathing resulted in respiratory acidosis because the pH fell below 7.35. Our min pH was at 7.25. But the Pco2 levels …show more content…

The hydrogen ions and sodium ions exchange places during the formation of urine. For every H+ that enters the urine, one Na ion is reabsorbed from the urine into the blood and is then conserved. Then for every H+ ion that is emitted and Na ion that is saved, one bicarbonate ion is reabsorbed into the blood. When we lowered the Pco2 there was a decreased concentration of ions in the urine, but elevated HCO3 in the urine. This means that all the ions are now in the blood. This can be recognized as hyperventilation/alkalosis, because of the depletion of carbon dioxide. This results in an elevated blood pH. When we raised the Pco2, H+ elevated and the HCO3 decreased. Since there are increased ions in the urine, this means that the ions are being secreted. This can be recognized as acidosis, since there was a lower than normal blood

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