Hippocampus Integrates

When diagnosed with probable Alzheimer's disease the effect of plagues and tangles are now starting to be seen in many ways including, memory loss in which individuals may have trouble learning new information and consistently ask the same question multiple times, along with disorientation which is an indication that the Hippocampus is being affected by plagues and tangles. The Hippocampus is usually one of the first areas affected by the disease (Brayne, 2014).
Once the disease spreads to the Hypothalamus some of the symptoms that may become evident are functions that are effected by different hormones such as, hunger, sleep, temperature regulation, sex drive and mood swings (Baloyannis, Mavroudis, Mitilineos, Baloyannis & Costa, 2014). Finally, the amygdala which is in the same area as the hippocampus and hypothalamus. The amygdala is mainly controls the emotions and as such damage or deterioration of the amygdala such as experienced with the onset of Alzheimers disease can lead to emotional instability which can induce effects such as anxiety, sadness, stress, anger and paranoia (LOOI & SACHDEV, 2000) found that at least eighty percent of Alzheimer's patients experience symptoms that can be directly related to deterioration of the amygdala. After these symptoms have developed the disease

moves on to other brain areas.

The thalamus is deep inside the brain, just above the brain stem. It is responsible for relaying motor and sensory signals to the cerebral cortex. It's also linked to the regulation of sleep and consciousness. Plus, it plays a role in controlling the motor systems of the brain responsible for voluntary bodily movement and coordination (Vishnu, 2013)

The temporal lobe is located to the side of the brain it is responsible for processing sensory information, long-term memory, and language skills. The plagues and tangles that develop in the temporal lobe produced by Alzheimer’s can result in the inability to articulate ideas and coherently express what is going on around them, as well as having impaired linguistic skills for example saying one thing but meaning another. It is also quite common for Alzheimer’s patients with damage in the temporal lobes to have perceived visual and auditory hallucinations. Moreover, the deterioration that Alzheimer's imposes on the temporal lobe inhibits the ability to recognize what should be familiar places, objects, and people (de Leeuw, Korf, Barkhof & Scheltens, 2006). When Alzheimers has advanced to its later stages, recalling personal information becomes severely compromised.

The occipital lobe is the area of the brain located at the back of the head and its primary purpose is to make sense of what our eyes are seeing. This region of the brain is not always affected by Alzheimer's disease, but when it is, the result can be difficulty in recognizing everyday objects, such as clothing, a toilet, or a bathtub. (Vecchio et al., 2013)

The frontal lobe is located at the front of the brain and is responsible for logical thought, regulating behaviours, complex planning, and learning.

Alzheimer’s disease gradually and continually damages the frontal lobe as the disease advances. As such, complex tasks such as driving, cooking, or advanced planning may become severely impaired by Alzheimer’s. Moreover, the impairment Alzheimer’s inflicts upon the frontal lobe may also result in the loss of motivation or drive, subsequently resulting in sluggishness and laziness. The damage that is caused by Alzheimer’s disease can create an ongoing loss of inhibition and impulsive behavior (George, Whitfield & Walker,

2013).

The parietal lobe is located at the top of the brain and is mostly responsible for assimilating and processing sensory information. Tasks such as reading, writing, mathematics, and spatial navigation are normally managed by the parietal lobe. Alzheimer’s systematically damages the parietal lobe during the various progressions of the disease. Symptoms such as the worsening of reading and writing skills, the inability to accurately locate items within a three-dimensional space, or properly gauge distance occur with increasing rate of occurrences and severity as Alzheimer’s intensifies in the parietal lobe (Gao, Li & Feng, 2011).

If you are suffering from Alzheimer’s disease it means that you are experiencing the gradual loss of your cognitive abilities. You will find that your memory becomes poorer as the condition progresses. Your ability to reason and make sense of things will not be as good as it used to be.
Yet, and despite in-depth research worldwide, there is no cure for Alzheimer’s disease. Also, it is a chronic (long-lasting) condition where symptoms gradually become more severe as the year's pass.

Extensive research has been carried out on Alzheimer’s disease based on its etiologic, pathogeny, epidemiology, heredity, mannerism, medication, psychology and the effects it exemplifies on the victims who are considerably older people. Accordingly, the considerable effects of the disease has led to physicians and medical researchers alike to deduce innovative methods that assist in combating the ailment through the principles of prevention and cure. Overtime, Alzheimer’s disease has continued baffling the findings and conclusions of the research and the strenuous efforts exuded by physicians and researchers. Regardless of the tenacity of the illness on aging people, innovations in medical technology have made it possible to mitigate the disease and the risks associated with it by providing methods and procedures that are effective and efficient in waging and prolonging the war against Alzheimer’s disease.

In conclusion, research on Alzheimer’s disease has paved the way for understanding and developing the pathology and treatment of the disease among aged people. Nevertheless, research in the ailment has also been conducted in the areas that involve prevention of the disease to prevent further financial and emotional distress on health care providers and family alike. Based on research evidence, preventive interventions have been deduced that restrict or delay the inception of Alzheimer’s disease. However, such interventions are either too complex to articulate or prevent the onset of the disease.