Arterial Pressure Lab Report

Introduction : The heart provides the body with all the nutrients it needs to preform its daily functions. Through systolic and diastolic pressures, the heart pumps blood to the the lungs in order to pick up oxygen and then to parts of the body. Pressure is created when the heart ejects blood from its chambers. That pressure is called the mean arterial pressure (MAP) and it is dependent on two variables: cardio output (CO) and total pressure resistance (TPR). Dr. Klabunde states, “In practice, MAP is not determined by knowing the CO and TPR, but rather by direct or indirect measurements of arterial pressure,” (2016). In order to test how MAP is affected by exercise, my partner and I developed an experiment. We first started with an obvious

The values for MAP were calculated by finding the pulse pressure, which is systolic pressure minus diastolic pressure. That number was then divide by 3 and added to the diastolic pressure. The data table revealed a 19% increase after level 1 (two minutes) and a 43% increase after level 2 (four minutes) for Josh. My level 1 and level 2 showed a 11% and a 21% respectively. Our data showed an increase in MAP as exercise increased. The second value calculated was CO. This was achieved by taking the heart rate multiplied by the stroke volume. Stroke volume did not to be calculated. The resting stroke volume is 70ml/beat and increased to 84ml/beat due to the fact that Josh and I are both relatively active people. Josh’s data provided a 41% increase at level 1 and 52% increase at level 2. My CO values increased by 25% for level 1 and 63% for level two. The data altogether reported and increase of CO with the increase of exercise. The final calculated value was TPR. TPR could not be directly measure by blood pressure or heart rate, but it could be found by taking the MAP values at each interval and diving it by the CO. Josh’s vitals showed a 16% and 6% decrease in TPR values. My values supported Josh’s as I had an 11% decrease at level one and a 26% decrease at level 2. Our vitals showed a decrease in TPR as exercise increased