Alzheimer's Disease Essay

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Alzheimer’s disease is the leading neurodegenerative disease in elderly adults. It affects more than 30 million people in the world (1). There are a few major markers behind Alzheimer’s disease. These include amyloid β plaque, oxidative stress, and inflammation. A potential target for the treatment of Alzheimer’s disease are the processes involved in the synthesis, transport, and function of retinoids.
Retinoids are vitamin A derivatives. They help to regulate differentiation and cell proliferation. Higher expression of retinoid signaling takes place in neuronal plasticity areas such as the hippocampus, retrosplenial, and prefrontal cortex (2). Retinoid signaling in the brain helps to control the brain functions. These include neurotransmitter release, neuronal differentiation, neurite growth, and long term potentiation (3). Retinoids are mediated by nuclear receptors (4). These nuclear receptors include retinoic acid receptors α, β, γ, and retinoid x receptors α, β, and γ.
Retinoids affect many different pathways. Some of the same pathways that retinoids affect are also affected when a person has Alzheimer’s disease. Retinoids regulate the expression receptors, neurotransmitter transporters, cell surface receptors, gene encoding enzymes, transcription factors, and neuropeptide hormones (3). A decrease in retinoic acid signaling may be associated with Alzheimer’s disease because it affects many different pathophysiological pathways (5). These pathways include amyloid β, neurotransmission, inflammation, and neurogenesis (3). Retinoids also regulate a few of the genes for neurons that contain recognition sequences for the retinoid receptor proteins.
One marker of Alzheimer’s disease are amyloid plaques. These plaques are extrac...

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...inoic acid is a possible way to decrease the oxidative stress levels in the brain.
Conclusion:
Retinoids can be used as a therapy to prevent Alzheimer’s disease. Vitamin A has an effect on many of the metabolic markers of Alzheimer’s disease. These include inflammation, oxidative stress, and amyloid β in the extracellular. Targeting the receptors for these processes may slow down or reverse Alzheimer’s disease.
The research into the effect Vitamin A has on Alzheimer’s disease needs to be furthered. One avenue to further it would be examining retinoic acids in vivo. This would allow the retinoid mechanisms of action and their receptors in Alzheimer’s disease to be better understood. Another method to further research would be to study the affect retinoic acid has on amyloid β in Alzheimer’s disease using different techniques, such as histopathological observation.

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