Sudden Infant Death Syndrome (SIDS)
Sudden Infant Death Syndrome (SIDS) or "crib death" is an abrupt and inexplicable death of an apparently healthy infant. Most of the cases involve infants from ages 1-12 months, and the event occurs during the night. Various theories have been postulated from research results but without consistency of the etiology. Since the death is sudden, prior diagnostic criteria or patterns are not available for correlation, although some near-miss infants have been followed. A number of possibilities have been documented in current literature, to include beta-endorphin changes, abnormal temperature regulation, pineal abnormalities, carotid body irregularities, lead poisoning, elevated fetal hemoglobin, brainstem immaturity, and cerebral hypoperfusion. The following is an overview of these pathologies in their relation to Sudden Infant Death Syndrome.
As with most physiological processes, several intermediate steps can lead to a certain event, thus making the mechanism more controlled. However, as more steps that are required, there arises a greater number of possible problems. SIDS is no exception. Most literature supports the view that victims of SIDS suffer a failure of the automatic control of respiration, producing periodic apnea and eventually death.
Neural control of respiration involves three anatomical structures (Armstrong et al., 1982~. The first is the motor system, which contains the neurons which initiate and maintain respiration. These include the dorsal motor nucleus of the vague, the nucleus tractus solitarius, the nucleus ambiguous, the nucleus retro-ambiguous, the reticulo-spinal tracts in the anterior and lateral columns and the anterior horn cells of the cervical and thora...
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...16:1122-1126, 1978.
Koceard-Varo, G. The physiological role of the pineal gland as the masterswitch of life, turning on at birth breathing and geared to it the function of the autonomic nervous system. The cause of SIDS examined in this context. Medical Hypothesis, 34:122-126, 1991.
Myer, E., Morris, D., et. al. Increased cerebrospinal fluid beta-endorphin immunoreactivity in infants with apnea and in siblings of victims of Sudden Infant Death Syndrome. J. Pedia., 111:660-666, 1987.
Quattrochi, J., McBride, P., and Yates, A. Brainstem immaturity in Sudden Infant Death Syndrome: A quantitative rapid Golgi study of dendritic spines in 95 infants. Brain Research, 325:39-48, 1985.
Takashima, S., Armstrong, D., Becker, L., et. al. Cerebral hypoperfusion in the Sudden Infant Death Syndrome? Brainstem gliosis and vasculature. Ann. Neurol., 4:257-262, 1978.
The seventeenth century was a time of great change in colonial America. Virginia, the first colony in the Chesapeake region, was established in 1624. Plymouth, the first colony in New England, was established in 1620. These two regions developed in distinct ways, but were intertwined because of their ties to England. The Chesapeake colonies were established for economic reasons, as the Virginia Company of London looked to mass-produce cash crops in the new world. The New England colonies, however, were created to be a religious haven for those who opposed the English church. Both regions developed economic and political systems that catered to the desires of the respective populations and the geographical conditions.
During the late 16th century and into the 17th century two colonies emerged from England. The two colonies were called the Chesapeake and New England colonies. Even though the two areas were govern by the English, the colonies had similarities as well as differences. The Chesapeake and New England colonies grew into obviously distinct establishments. Difference in colonial motivation, religious, political structures, socio-economic, and race relation, were responsible for molding the territories.
New England was a refuge for religious separatists leaving England, while people who immigrated to the Chesapeake region had no religious motives. As a result, New England formed a much more religious society then the Chesapeake region. John Winthrop states that their goal was to form "a city upon a hill", which represented a "pure" community, where Christianity would be pursued in the most correct manner. Both the Pilgrims and the Puritans were very religious people. In both cases, the local government was controlled by the same people who controlled the church, and the bible was the basis for all laws and regulations. From the Article of Agreement, Springfield, Massachusetts it is ...
Chesapeake and New England both ended up prospering in the colonial era, even with the widely different institutions and opinions they each held. The forces of motives for founding the colonies, geography, the settlers themselves influenced the contrast. However, in the next century New England and Chesapeake would discover the forces of freedom and liberty would lead them to find a common ground--that of breaking free from Great Britain in the American Revolution.
The first reason why the difference in development between New England and the Chesapeake region occurred was because they were founded for different purposes. The primary reason for the settlement of New England was to create a safe haven for the Puritans of England where they could freely express their religion. The Puritans believed that it was their responsibility and God’s expectation that they create moral, Christian communities. John Winthrop reflects this in document A when he says that their failure would “open the mouths of enemies to speak evil of…God.” The Chesapeake settlers
The New England Colonies purposes were different than the southern colonies. In the north most of the colonies were settled for religious freedom, whereas in the south they settled for profit. With having different motives for their societies, colonization became very different. The Chesapeake region originally were wasting all their time looking for gold, for money, and didn't come prepared for survival in the unknown, New World. The pilgrims, who were separatist came to the new world and were ready to “set up camp” as they were there in search to create a society where they could live by their faith without the fear of the monarch disapproving. When Maryland was
We know babies die from SIDS and they have been looking high and low for a cause. Everyone seems to want a neat and tidy answer to what has happened to these babies, and I understand why. I believe co-sleeping has been given a bad reputation because people need something to blame and not based on actual scientific evidence. Dr. William Sears suggests that, “In those infants at risk for SIDS, natural mothering [unrestricted breastfeeding and sharing sleep with baby] will lower the risk of SIDS” (Sears, "Cosleeping and Biological Imperatives").... ...
In the time period leading up to 1700, American history was a time of tremendous settlement and establishment of colonies across the nation. In determining how the colonies were to be created, the settlers had to question how long they were going to live at these locations; as well as, which places were flowing with the resources and materials they were searching for. In this case, English settlers founded the New England and Chesapeake regions in the early 17th century; however, the two regions became different from each other as time passed, for each became distinct colonies by 1700. Although the English settled both New England and the Chesapeake region, these two regions differed in development because though both were ruled by an English
In the early 1600’s there was the development of New England and Chesapeake Bay colonies, and even though they were both settled by people mainly of the English origin by 1700 they became very two distinct societies. As the two colonies evolved, they developed contrasting economies, societies and institutions.
Person, A. & Mintz, M., (2006), Anatomy and Physiology of the Respiratory Tract, Disorders of the Respiratory Tract, pp. 11-17, New Jersey: Human Press Inc.
This study is a clinical trial that aims to find out the effect of massage on behavioral state of neonates with respiratory distress syndrome. The participants were 45 neonates who hospitalized in neonatal intensive care unit of Afzalipour hospital in Kerman. Parental consent was obtained for research participation. The inclusion criteria included all infants born with respiratory distress syndrome, less than 36 weeks gestational age and without of any the following conditions: contraindication of touch, skin problems, hyperbilirubinemia, anemia, respirators, chest tube, addicted mother, congenital and central nervous system disease. Infants entered the massage protocol during the second day after starting enteral feeding, because the initiation of enteral feeding means that the infants in physiologically stable [12]. The researcher determined if infants met the study criteria. After initial assessment, the infants were entered to the group. The infants received 45 minute periods of massage intervention per day for 5 days. Each infant received tactile/kinesthetic stimulation, 15 minute periods at the beginning of three consecutive hours. Each massage always started at approximately 30 minutes after afternoon feeding and provided by one or two trained nurses. The 15 minute stimulation sessions consist of 3 standardized 5 minute phases. Tactile stimulation was given during the first and third phases, and kinesthetic stimulation was given during the middle phase. For the tactile stimulation, the neonate was placed in a prone position. After thorough hand scrubbing, the person providing stimulation placed the palms of her warmed hands on the infant’s body through the isolate portholes. Then She gently stroked with her hands for five ...
Sudden unexpected death in epilepsy (SUDEP) is the most common cause of death directly related to epilepsy, and most commonly occurs in people with chronic epilepsy (Surges, 2009). Information provided to people with epilepsy should take account of the small but precise risk of SUDEP.[3] SUDEP seems to occur more commonly during sleep and more often affects young adults with medically uncontrollable epilepsy (especially tonic-clonic seizures), those with neurological comorbidity, and patients receiving antiepileptic drug polytherapy.[7] The risk of SUDEP can be diminshed by optimizing seizure control and being aware of the potential consequences of nocturnal seizures.[3]
...ble. Although this is so, certain cases have indicated that some patients’ brains appeared lifeless, when they are in a coma, but are not completely brain dead, therefore the ventilation and persistence of their family has been beneficial to them. Recent cases also indicate that foetuses can be kept alive in their mother’s womb using ventilation if the mother has been declared brain dead.
The Problem people have with philosophy is the lack of definite answers. This is not to say that philosophy has never produced any. Russell
The ANS is positioned just below the medulla oblongata in the lower brainstem. The medulla is responsible for many major functions, such as respiration, cardiac regulation, vasomotor activity, and reflex actions; which include coughing, sneezing, vomiting, and swallowing. The input is received by the hypothalamus, which is located right above the