Rheumatic Fever Essay

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Rheumatic fever is a disease that can occur following bacterial infection with Group A Streptococcus. Predisposing infections also include strep throat tonsillitis and skin infections, such as impetigo, caused by Streptococcus pyogenes. Overall, rheumatic fever is rare in Australia; however, the rate of rheumatic fever amongst Indigenous Australians is much higher. Rheumatic fever is a serious condition that can lead to long-term complications, such as rheumatic heart disease.
Causes & Pathophysiology
Most commonly, rheumatic fever occurs following a tonsillitis throat infection with Group A Streptococcus. It can also follow skin infections caused by the same organisms; this is especially the case for many indigenous Australians. The symptoms of rheumatic fever can take up to one month to develop following the initial infection. Rheumatic fever is an acute infection with an interesting pathogenesis. It is the result of the immune system producing antibodies to fight off the group A streptococcus infection. Rheumatic fever is a type-II hypersensitivity reaction, which means that the antibodies produced by the immune system bind to the antigens on the body’s own tissues and cause a response.
Group A Streptococcus organisms have 3 predominant virulence factors which help to establish an infection:
1. M proteins: M proteins are found on the surface of the organism and protect it against phagocytosis. The M proteins prevent the attachment of complement proteins to the cell. Complement proteins which are attached to the bacterium “tag” it for destruction by phagocytic cells, such as neutrophils and macrophages, in a process called opsonisation. By inhibiting this process, the M protein allows the group A streptococcus to survive longer...

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The blood tests performed when post-streptococcal glomerulonephritis is suspected are similar to those discussed above with rheumatic fever. In this condition, blood pressure is often high and should also be measured. A sample of urine may also be assessed to assist in making the diagnosis.
The main treatment for post-streptococcal glomerulonephritis involves a course of antibiotics (usually penicillin) to kill off the remaining Group A Streptococcus. Diuretics may also be prescribed to reduce the amount of fluid in the body and hence the amount of swelling. These may also help to control blood pressure, and in some instances other blood pressure medications may also be used. Unlike rheumatic fever, preventative antibiotic injections are not given to patients with post-streptococcal glomerulonephritis because it is uncommon to have the infection more than once.

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