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Migraine pathophysiology essay
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Chief Complaint
Migraine headaches.
History
Patient is a 19-year-old right-handed white male who presents with his mother for evaluation of frequent headaches. He did not have headaches prior to two grade 1 concussions while playing football in 2012. At that time, he had a normal MRI. He has been having headaches since. He did see Kent Logan, MD in 2012, at which point he was describing weekly headaches with photophobia, phonophobia, and nausea. At that time, according to Dr. Logan's notes, there was no aura with his headaches. He noted that trying one of his mother's Imitrex helped with the headache, so he was given a prescription for 50 mg. He was also diagnosed as having a whiplash injury, at that time. He did undergo physical therapy for his neck. He also was complaining of some short-term memory problems at that time, but neuropsychology testing was negative. He has not followed up with Dr. Logan since then. More recently, his headaches have been increasing in frequency. They are located in the left retrobulbar and super orbital area, but then will spread throughout the left side of the head and then bilaterally. The pain is steady when it is milder, but throbbing when it is worse, and it is worsened with exertion. There is photophobia, phonophobia, osmophobia, nausea. They can last one to two hours. Most often, he does have
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a scintillating scotoma with them. Precipitants include odors, sun, possibly bouncing around in the tractor, prolonged reading or computer work and having to take tests. He was given 25 mg of Imitrex by his PCP to hold him over. Over the past several months, he has been using Aleve or Motrin very frequently. His frequency is two to three headaches a week and it has been like this for about four months. Last year, he was having maybe two or three headaches a month. He also has a history of vasovagal syncope, not always temporally related to a headache. It occurs when he is upright, never when he seated or lying. There is no tonic or clonic motion. There is no tongue biting, bowel or bladder incontinence. He has no other symptoms that sound like nocturnal, generalized, or partial seizures. He has not had any cardiac work up or tilt table testing. He does have panic attacks that are related, and anxiety that are related to these episodes of vasovagal syncope. Also, his mother notes that when he starts to get a migraine or migraine aura, he becomes anxious about it. His anxiety regarding his syncope has led to a significant change in his day-to-day living in that currently he no longer flies alone to visit his father in Minnesota. The vasovagal syncope and anxiety are significantly affecting his quality of life. Review of Systems Syncope, neck pain, poor sleep.
Social History
He does not smoke. He does not drink alcohol. He is at Great Bay studying liberal arts.
Family History
Mother and maternal grandmother with migraines, breast cancer in the family, diabetes, SLE, fibromyalgia.
Past Medical History
He does have vasovagal syncope 15 to 20 times a year or more.
Panic attacks three to four times a week.
He did have Burkitt's lymphoma at three years old and underwent chemotherapy. He was told that he would have cardiac issues after that, but follow up EKGs have been normal.
Allergies
Cephalosporins, amoxicillin,
Augmentin. Medications Imitrex 25 mg, lorazepam 0.5 mg. Examination Constitutional Weight 180 pounds. Height 5'10". Respirations 12. Pulse 69. General He is in no obvious distress. Cardiovascular Carotids reveal no bruits. Mental status He is oriented x3, alert, cooperative. Good short-term, long-term and intermediate memory. No aphasia. Normal fund of knowledge. Normal attention and concentration. Cranial Nerves The fundi were benign. Visual fields full to confrontation. Extraocular muscles intact. PERRLADC. Normal facial symmetry, sensation, and movement. Tongue and uvula were midline. Normal auditory acuity. Normal shoulder shrug. Motor Was 5/5. No fix or drift. Normal tone all four extremities. Sensory Was intact to primary modalities with no extinction on double simultaneous stimulation. Cerebellar Revealed good finger-to-nose, heel-to-shin and rapid alternating motion. Gait Normal. Negative Romberg. DTRs 2+ throughout. Toes are downgoing. Impression This is an individual who does have migraine with aura, and they are frequent enough for prophylaxis. Currently, he may also be suffering from a component of medication overuse headache. He also has frequent vasovagal syncope, which is resulting in anxiety and panic attacks and significantly adversely affecting his quality of life. He also has some neck pain, which he thinks may be precipitating some of the migraines. Plan My plan right now is to start him on amitriptyline, titrating to 25 mg at night. This is for his migraine prophylaxis. It also may help with the cervicalgia and certainly will help to regulate his sleep. It may also have a mild anti-anxiety component to it. I am giving him Imitrex 50 mg to use p.r.n. He is going to keep a headache diary. I am going to do an EEG just to rule out any possible seizure-like activity, after which I will discuss with the mother and patient whether or not to set him up with a cardiologist for a full cardiac evaluation and possibly tilt table test, considering his Burkitt's lymphoma, and a being told as a child that he may have cardiac issues. I will see him in followup in one month.
Trigeminal Autonomic Cephalalgias (TACs) are highly interesting to me: This group of unilateral, excruciating primary headaches is accompanied by ipsilateral cranial autonomic symptoms and comprises of three major forms:
and they were different from most headaches he had in the past. He would forget things, and then forget more often. He said he had a vague feeling that something just wasn’t right. One day he had a seizure while he was at work.
In his college years it was pretty hard for him to find a date. So, he stayed at home and
In 2005 Dr. Bennet Omalu first discovered CTE In the Brain Of the retired player Mike webster, after he passed in 2002, at the age of 50. However CTE has been identified, there is no way to diagnose it in living individuals. In a study done by the National Institutes of Health, and the Concussion Research Funding, they came to the conclusion that, “Current tests cannot reliably identify concussions, and no technique reliably differentiates individuals who will recover quickly, suffer long-term symptoms, or develop chronic traumatic encephalopathy (CTE)” (NIH, NFL, and Concussion Research Funding). Therefore, currently there is no way to treat or identify whether or not a person has CTE officially until they have died. Doctors only way of identifying if a person has CTE is to diagnose based off their symptoms. CTE symptoms vary based on the severity of the case, However according to researchers at the CTE Center at Boston University School of Medicine; “CTE is associated with “memory loss, confusion, impaired judgment, impulse-control problems, aggression, depression, and, eventually, progressive dementia” (Karaim). Theses Symptoms have a dramatic effect on the everyday lives of the people that have CTE. With nearly anywhere from 1.6 million to 3.8 million concussions occur each year, leading professional athletes and
I know my subject by his first name only, Maynard, by listening to his frequent conversations with his close friend Rolando I have discovered they enjoy talking about alcoholic beverages, movies, and video games among other things. He is of average height and a slim build with frizzy short black hair and wears glasses.
in all but one of his subjects. He does not like to talk about his
He stated to study literature. He took creative-writing course at Chico State College on 1958.
This condition can be treated various ways depending on “the type and frequency of arrhythmias, associated symptoms…, and the presence of structural heart disease” (Cleveland Clinic, 2014). Some patients may not need treatment at all, since they show not symptoms, since sometimes this condition can be naturally eliminated over the first year of life, but may still be required to have regular schedule appointments with the physician so the patients can be monitored. If symptoms are prevalent, the different treatment methods include a pacemaker, defibrillator, surgery, and medicine.
When he fish school he go on to collar and it was call massachusetts. He was smart guy he got only class room to techecher
His initial blood pressure was 113/60 mmHg, her heart rate was 50 beats/min, and his oxygen saturation was 100%. His Glasgow Coma Scale was 8. His physical exam was also remarkable for dry mucous membranes, and distant heart sounds, in addition to weak lower extremity pulses and non-pitting edema. No thyroid goiter was palpable.
He had seizures that were mostly nocturnal since about age 16. He is a little uncertain about the course of those later but he did continue to have seizures of some kind or other up until 2015. That might have been nocturnal and it might have been alcohol withdrawal related, he is not clear. For a period of time, he was on gabapentin through the Native American Clinic in Minneapolis for seizure control and for left lower quadrant pain related to an old knife wound. He has also been taking amitriptyline for migraine related to several TBI. He has had several MVAs and assaults resulting
Today is a good day. Today there is only mild throbbing in my right temple. Feels like a small ice pick jabbed in my head. Unlike a bad day when it feels as if I am giving birth through my head just after someone beat my skull and neck with a baseball bat. This is a small example of how it feels to live with migraines. It was an ordinary day at work back in 1987 when I received my first visit from the migraine. The day was unforgettable. I was twenty years old. Out of nowhere, my peripheral vision became blurry. It was hard to see. Shortly after, I began seeing black spots. I was scared. I thought I was going blind. Nausea soon followed. I told my boss I was not feeling well and needed to go home. The twenty-two-mile journey
Per the medical report dated 05/17/16, the patient reported bilateral hand pain with numbness and tingling for the past 7 years or so, worsening, right greater than the left side. There is some degree of numbness/tingling on the right hand at all times. She has difficulty sleeping at night due to pain and driving, doing her hair/make up or holding objects worsen her symptoms. She has tried wearing splints that they worsened the discomfort. She denies any history of steroid injections. On examination, Tinel’s, Durkan’s and Phalen’s tests are positive bilaterally. There is bilateral thenar weakness.
Cardiovascular System: He does not experience any chest pain or palpitation. He does not have dyspnea or leg swelling.
This 70-year-old patient who presented after what appeared to be a syncopal episode. According to her grandson the patient had a change in mental status at home she was cooking, felt dizzy, went down to sit down and slumped over. It was estimated that she was out with loss of consciousness for about 15 minutes. She was subquently brought to the emergency room. When she did awake she was somewhat confused. She has a prior history of a stroke in 2014, dyslipidemia and hypertension. Initial evaluation her blood pressure was 135/96. Her heart rate was 60. Her EKG had sinus rhythm with no significant abnormalities. Her CT of her head showed no acute mass or infarct. There was also suspicion of urinary tract infection and the patient was