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Hypertension etiology and pathophysiology
Hypertension etiology/pathophysiology
Pathophysiology of hypertension
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Recommended: Hypertension etiology and pathophysiology
The patient that stimulated the interest in this subject was a 57-year-old male admitted with an acute exacerbation of COPD. The patient also had a history of hypertension, pulmonary embolism, and deep vein thrombosis. The patient is a smoker as well as his family. He has had many problems with trying smoking cessation for years. The patient also has a history of depression and anxiety. The patient has been on many medications to control his hypertension, but it is still out of the normal range. This man was in great need of nonpharmacological intervention and education.
The focus of this paper will be presenting a general overview of the etiology of, global impact of, and compliance with treatment of hypertension. It will also cover the nonpharmacological interventions aimed at managing hypertension. Finally it will explore caring for the psychosocial needs of the patient and how this can help to treat and possibly prevent hypertension.
One of the problems with essential hypertension is that it has no clear etiology. In one of the articles covered in this paper the essential mechanisms of hypertension are reviewed. The three systems that affect blood pressure I will cover in this paper are the kidneys, the immune system, and the nervous system.
The kidneys play a vital role in the control of blood pressure. They do this by controlling the sodium retention and excretion, which in turn controls blood volume and thus blood pressure. In one study, the kidneys of hypertensive rats were exchanged with other rats and it was found that the hypertension shifted to the new rat that received the hypertensive kidney (Coffman, 2011). This shows the importance of the kidney in hypertensive patients.
The article also revealed that an...
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...ent of their diseases.
Works Cited
Coffman, T. (2011). Under pressure: the search fot the essential mechanisms of hypertension. Nature Magazine , 1402-1409.
DeSimone, M. E., & Crowe, A. (2008). Nonpharmacological approaches in the management of hypertension. Journal of the American Academy of Nurse Practitioners , 189-196.
Hildingh, C. (2009). The association among hypertension and reduced psychological well-being, anxiety and sleep disturbances: a population study. The Scandinavian Journal of Caring Sciences , 366-371.
Lee, D. E., & Cooper, R. S. (2009). Recommendations for Global Hypertension Monitoring and Prevention. Current Hypertension Reports , 444-449.
Osamor, P. E., & Owum, B. E. (2011). Factors Associated with Treatment Compliance in Hypertension in Southwest Nigeria. Journal of Health, Population, and Nutrition , 619-628.
Vicki is a 42-year-old African American woman who was diagnosed with Hypertension a month ago. She has been married to her high school sweetheart for the past 20 years. She is self-employed and runs a successful insurance agency. Her work requires frequent travel and Vicki often has to eat at fast food restaurants for most of her meals. A poor diet that is high in salt and fat and low in nutrients for the body and stress from her job are contributing factors of Vicki’s diagnosis of hypertension. This paper will discuss the diagnostic testing, Complementary and Alternative Medicine treatments, the prognosis for hypertension, appropriate treatment for Vicki, patient education, and potential barriers to therapy that Vicki may experience.
As early as the 1800s clinicians began to take a closer look at elevated blood pressure levels, they soon found high correlation between hypertension, stroke, and other heart diseases. They also established that high levels of blood pressure effected both privilege and underprivileged, and within the years they have noted the disease have become more prevalent in the African American culture. Long term studies, such as randomized controlled trial studies, unveiled
My PICOT question was developed after reading the case study about a patient named Vincent Brody who had for the last 50 years of his life consumed 40 cigarettes a day and despite his diagnosis of Chronic obstructive pulmonary disease (COPD) and a recent episode of exacerbation of his condition that required him to be admitted to hospital he was continuing to smoke.
Client Profile: Lane Bronson is a 55 year old male with a history of angina, hypertension, Type 2 diabetes, COPD, and sleep apnea. He comes to the physican’s office complaining of worsening shortness of breath. His skin tone is grey, and his angina is worsening. Previously stable, he now does not get relief from rest or nitroglycerin. The physician called 911 and had Mr. Bronson directly admitted to the hospital.
High blood pressure is called the “silent killer” because it often has no warning signs or symptoms, and many people don’t know they have it. For most patients, high blood pressure is found when they visit their health care provider or have it checked elsewhere. Because there are no symptoms, people can develop heart disease and kidney problems without knowing they have high blood pressure. Some people may experience: bad headache, mild dizziness, and blurry vision. Traditionally, diagnosis of high blood pressure (BP) has relied on consecutive checks of clinic BP over a 2 to 3 month period, with hypertension confirmed if BP remains persistently raised over 140/90 mmHg. This method of diagnosis has significant limitations because the BP measured for an individual patient in a clinic setting may not reflect their BP in day-to-day life. The main concern is that as a result of the “white coat syndrome”, hypertension may be over-diagnosed when checked in the clinic setting; resulting ...
The causes of hypertension are unknown. However; hypertension can be classified into two categories primary and secondary. Primary (essential) hypertension is increas...
Hypertension is a developing problem worldwide,associated with an increased risk of cardiovascular morbidity and mortality. In 2020, the world population will be approximately 7.8 billion people, and there will be 1 billon people who may be affected by hypertension (Tomson & Lip, 2005). One in three adults in the United States has high blood pressure. According to the report “Health, United States, 2010 with Special Feature on Death and Dying,” the prevalence of hypertension among adults 20 years old or older increased from 24% to 32% during 1988-1994 and 2005-2008. The African-American population has a higher prevalence of this health condition than white Americans do. In 2005 to 2008, the prevalence of hypertension among black males (41.4%) was more than 10%, compared to white males at 31.5% of the population. The hypertension rate of black females was more than twice that of white females. The death rate from hypertension among black males was 51.8 per 100,000, and among black females was 40.4 per 100,000; however, the morta...
Hypertension, also known as high blood pressure is a medical condition where the arteries are constantly experiencing high blood pressure due to the force exerted on the walls of the arteries as the heart pumps blood throughout the body.
" Journal of Clinical Hypertension 13.5 (2011): 351-56. EBSOHost.com - a. Web. The Web.
There are generally two categories of hypertension, primary hypertension and secondary hypertension. The causes of primary hypertension are usually unknown and it develops gradually over the years. As for secondary hypertension, the causes are mainly underlying other health problems and it develops in a sudden of time. (The Healthline Editorial Team 2013) First of all, hypertension can be related to renin-angiotensin-aldosterone system (RAAS). Renin is an enzyme secreted mainly via the juxtaglomerular apparatus of the kidney when the sodium retention and water retention are reduced in the blood. (Beevers, Lip, and O'Brien 2001) Renin will bind to a substrate which is angiotensinogen and form angiotensin I which is an inactive peptide. (Foëx and Sear 2004) After that, angiotensin I will be rapidly converted to an active peptide angiotensin II by angiotensin-converting enzyme (ACE) in lungs. As a result, vasoconstriction occurs due to presence of angiotensin II and this increases the blood pressure. Moreover, angiotensin II can stimulate the releasing of aldosterone by adrenal glands. Secretion of aldosterone raises the blood pressure by reabsorbing sodium salts and water to increase sodium and water retentions. (Beevers, Lip, and O'Brien 2001)
The trial consist of patient who were 35 years old or older with clinical diagnosis of mild to moderate COPD (confirmed FEV1/FVC LESS 70% and FEV1 % predicted normal value greater or equal to 50% and willingly to stop smoking. Participants has higher percentage of male and Caucasians populations, smoked for an average of 40 years also smoked 10 or more cigarettes per day over the past year. Participants treated with systematic steroids or hospitalized for COPD exacerbation during the 4 week period were excluded. Aslo excluded are participants with uncontrolled medical
WILLIAMS, P and POULTER NR et al (2004) Guidelines for management of hypertension: report of the fourth working party of the British Hypertension Society. British Hypertension Society, pp. 139-85
U (To maintain the patient’s confidentiality, pseudonyms will be used throughout the essay NMC (2004) and the clinical setting will remain anonymous.) was brought to the hospital by her daughter, due to breathlessness, decreased mobility, loss of appetite and recurrent cough productive of runny, translucent – yellowish sputum. Mrs. U is a chain smoker for 40 years.
The pathogenesis of hypertension is multifactorial and highly complex. There are several influential factors that control blood pressure (BP) for adequate tissue perfusion, such as vascular reactivity, circulating blood volume, blood viscosity, cardiac output, blood vessel elasticity, and neural stimulation. Increased peripheral resistance, vascular rigidity, and vascular responsiveness to stimuli are significant to the pathophysiology of hypertension. Sodium has a primary effect on blood pressure leading to excess circulating volume, affecting hypertrophy, contractility, and vascular resistance (Buttaro, Trybulski, Bailey, Sandberg-Cook, 2011).
Schnall, P., Landsbergis, P., Belkic, K., Warren, K., Schwartz, J., & Pickering, T. (1998). Findings In The Cornell University Ambulatory Blood Pressure Worksite Study: A Review. Psychosomatic Medicine, 60, 697.