Deep Vein Thrombosis Teaching Plan

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Recommended: Deep vein thrombosis pathology

Teaching Plan: Deep Vein Thrombosis
Rationale
The patient that this teaching plan is based on is a 55 year old female that came in having Deep Vein Thrombosis, and stomach pain. The patient has a history of diabetes and hypertension. She currently takes meds to treat these two diseases. She just recently found out that she has squamous cell carcinoma, and that it has become malignant. The patient had a swollen right calf, and she denied that she had pain in that leg. She was started on heparin therapy and sequential compression devices (SCDs) to keep more clots from forming. The patient’s PT/INR and aPTT came back abnormal possibly due to the patient having a DVT.
Outcomes
1. The patient will explain what DVT is by time of discharge.
2. The
Deep veins are located in the arms, legs, pelvis, and pulmonary system (Wipke-Tevis & Rich, 2014). Risk factors that are associated with a venous thromboembolism are venous stasis, endothelial damage and hypercoagulability of the blood. Pathophysiology behind a DVT is when platelets aggregate and fibrin capture the RBCs, WBCs, and platelets to form a clot. Common sites where thrombus is formed are valve cusps of veins, where venous stasis happens (Wipke-Tevis & Rich, 2014).
Some signs and symptoms of a DVT is that a patient may have unilateral leg edema, pain tenderness with palpation, paresthesia, and warm skin. When a DVT is not treated, a clot can travel to the lungs, and become a pulmonary embolism. Pulmonary embolisms are considered a medical emergency. The patient can display dyspnea, chest pain, sweating, fainting or coughing up blood. Venous thromboembolism (VTE) is used to describe DVT and PE because the two conditions are closely related (“Discharge Instructions for Deep,” 2017). The labs that are monitored are D-dimer, PT/INR, PTT or aPTT, platelets, hemoglobin, and hematocrit. The labs that are discussed in the sentence before were all abnormal. Patients are given anticoagulants for VTE. This patient was started on heparin therapy, but then was weaned off of that. She was prescribed Enoxaparin (Lovenox) which is a low molecular weight heparin that helps prevents a VTE from forming.

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