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Atrial fibrillation etiologypathophysiology
Atrial fibrillation etiologypathophysiology
Atrial fibrillation pathophysiology essay
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Atrioventricular node reentrant tachycardia
Other than atrial fibrillation and atrial flutter, this is the most common supraventricular tachycardia seen in practice.11 A large minority of adults (up to 40% in some cohorts) are born with 2 pathways that can conduct electricity in the AV node, rather than 1. Under the right conditions, AV node reentrant tachycardia (AVNRT) can be initiated by a premature atrial or ventricular beat. If the 2 pathways are able to sustain a stable circuit, the atrium and the ventricle are depolarized almost simultaneously. As a result, on the ECG, the P wave is not seen, is buried in the QRS complex, or is seen at the terminal portion of the QRS, typically as a pseudo-s (negative) wave in the inferior leads or a pseudo-r0 in lead V1. Because this tachyarrhythmia depends on the AV node, both vagal maneuvers and adenosine are potential acute treatment options. In the outpatient setting, if the patient is in a sustained supraventricular tachycardia, attempting vagal maneuvers is reasonable. These maneuvers should include bearing down,
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The ECG in this setting should have a typical right or left bundle branch block appearance. A typical example of this tachyarrhythmia is atrial flutter with 1:1 AV conduction.
Supraventricular tachycardia with atrioventricular conduction via an accessory pathway This entity is only (and rarely) seen in patients with Wolff-Parkinson-White syndrome. It involves conduction of the atrial signal antegrade down the accessory pathway and retrograde up the AV node. Because ventricular depolarization occurs cell to cell, rather than via the normal conduction system, the QRS is wide and mimics monomorphic ventricular tachycardia. This tachycardia is also known as antidromic AVRT.1 A prior ECG with a delta wave typical of Wolff-Parkinson-White syndrome is helpful for this
There are several different heart problems that show up as an abnormal EKG reading. For example, a heart block can occur when there is a delay in the signals coming from the SA node, AV node, or the Purkinje fibers. However, clinically the term heart block is used to refer to an AV block. This delays or completely stops communication between the atria and the ventricles. AV block is shown on the EKG as a delayed or prolonged PR interval. The P wave represents the activity in the atria, and the QRS complex represents ventricular activity. This is why the PR interval shows the signal delay from the AV node. There are three degrees of severity, and if the delay is greater than .2 seconds it is classified as first degree. Second degree is classified by several regularly spaced P waves before each QRS complex. Third degree can be shown by P waves that have no spacing relationship to the QRS complex. Another type of blockage is bundle branch block. This is caused by a blockage in the bundle of His, creating a delay in the electrical signals traveling down the bundle branches to reach the ventricles. This results in a slowed heart beat, or brachycardia. On an EKG reading this is shown as a prolonged QRS complex. A normal QRS is about .8-.12 seconds, and anything longer is considered bundle branch block. Another type of abnormal EKG reading is atrial fibrillation, when the atria contracts very quickly. On the EKG this is shown by no clear P waves, only many small fibrillating waves, and no PR interval to measure. This results in a rapid and irregular heartbeat. On the other hand, ventricular fibrillation is much more serious and can cause sudden death if not treated by electrical defibrillation.
According to the doctor, the patient might have developed congestive heart failure. Is it right-sided or
622 Y. When the AV node receives the signal, it fires and causes the ventricles to depolarize, this is known as the QRS Complex. The atria also repolarizes during this phase. Specifically in the QRS Complex, during the Q wave, the interventricular septum depolarizes, during the R wave, the main mass of the ventricles depolarizes, and during the S wave, the base of the heart, apex, depolarizes. After the QRS Complex, the S-T segment can be identified as a plateau in myocardial action potentials and is when the ventricles actually contract and pump out blood to the pulmonary and systemic circuits. The final phase of the heartbeat is the T wave and this is when the ventricles repolarize before the relax, ventricular diastole, EKG Video Notes and pg. 671 D. These phases represent the cardiac cycle, which is the time and events that occur from the beginning of one heartbeat to the beginning of the next heartbeat. In this lab, the first EKG that I took was my regular heartbeat during rest. In this recording, I was able to see the P wave, followed by the QRS Complex and the T wave as well. Everything looks pretty normal, but the T wave does go a little lower than normal and I believe this is due to the fact that I was diagnosed with sinus bradycardia
Stadler P, Hoch M, Radu I. Echocardiography in the horse with special regard to color-flow Doppler technique. Prakt Tierarzt 1995; 76: 1015
When a muscle contracts and relaxes without receiving signals from nerves it is known as myogenic. In the human body, the cardiac muscle is myogenic as this configuration of contractions controls the heartbeat. Within the wall of the right atrium is the sino-atrial node (SAN), which is where the process of the heartbeat begins. It directs consistent waves of electrical activity to the atrial walls, instigating the right and the left atria to contract at the same time. During this stage, the non conducting collagen tissue within the heart prevents the waves of electrical activity from being passed directly from the atria to the ventricles because if this were to happen, it would cause a backflow. Due to this barrier, The waves of electrical energy are directed from the SAN to the atrioventricular node (AVN) which is responsible for transferring the energy to the purkyne fibres in the right and left ventricle walls. Following this, there is a pause before the wave is passed on in order to assure the atria has emptied. After this delay, the walls of the right and left ventricles contract
It occurs because of repetitive electrical activity. This can occur in a patient with early or late heart failure, because there is damage to the heart tissue and the heart beats faster to try to supply the body with blood. Recommended treatment is elective cardioversion. Drugs used include an antidysrhythmic such as Mexitil or Sotalol (Ignatavicius &Workman, p. 728-729).
Two heart sounds are normally heard through a stethoscope on the chest wall, "lab" "dap". The first sound can be described as soft, but resonant, and longer then the second one. This sound is associated with the closure of AV valves (atrioventricular valves) at the beginning of systole. The second sound is louder and sharp. It is associated with closure of the pulmonary and aortic valves (semilunar valves) at the beginning of diastole. There is a pause between the each set of sounds. It is a period of total heat relaxation called quiescent period.
Atrial fibrillation (AF) is a cardiac arrhythmia. It is the most common arrhythmia and it has implications for patients and anaesthetists alike. The anaesthetist must take into consideration the physiological and pharmacological implications of this common arrhythmia.
Eisenmenger Syndrome (ES) is a heart defect that was first giving the name in 1897 (Fukushima, 2015). This syndrome happens when the birth defect is not treated before the lungs’ arteries become damaged. Eisenmenger Syndrome is named after Victor Eisenmenger a man who had a patient who showed symptoms such as, breathing complications and skin that was turning a bluish color. The autopsy of this patient lead him to discover a ventricular septal defect [VSD] (El-Chami, 2014), that causes a hole in the wall on the right and left ventricular. This is the defect that begins when signaling for pulmonary artery hypertension, which progresses into more advanced stages of ES. This birth defect eventually causes patients to have various
In a normal strip, one can clearly identify a P wave before every QRS complex, which is then followed by a T wave; in Atrial Fibrillation, the Sinoatrial node fires irregularly causing there to be no clear P wave and an irregular QRS complex (Ignatavicius & Workman, 2013). Basically, it means that the atria, the upper chambers of the heart, are contracting too quickly and no clear P wave is identified because of this ‘fibrillation’ (Ignatavicius & Workman, 2013). Clinical Manifestations and Pathophysiology A normal heart rhythm begins at the sinoatrial node and follows the heart's conduction pathway without any problems. Typically the sinoatrial node fires between 60-100 times per minute (Ignatavicius & Workman, 2013).
The study of cardio physiology was broken up into five distinct parts all centering on the cardiovascular system. The first lab was utilization of the electrocardiogram (ECG). This studied the electrical activities of the heart by placing electrodes on different parts of the skin. This results in a graph on calibrated paper of these activities. These graphs are useful in the diagnosis of heart disease and heart abnormalities. Alongside natural heart abnormalities are those induced by chemical substances. The electrocardiogram is useful in showing how these chemicals adjust the electrical impulses that it induces.
The second beat is the semilunar valve opening to allow blood into the aorta or pulmonary trunk. The cardiac cycle is composed of five stages. These stages are atrial systole, early ventricular systole, late ventricular systole, early ventricular diastole, and late ventricular diastole. In order for atrial systole to occur, the blood that has been flowing between the atrium and ventricle via the opened atrioventricular valves must be deposited into the ventricles. The SA node is responsible for the contraction of the atrial myocardium.
The defibrillator monitors the heart and automatically gives electric shocks before Arrhythmia causes permanent damage. If the person does not have a defibrillator a strong electric shock will be given. The cause of Tachycardis is by something not functioning properly in the atria. They are sometimes stimulated by anxiety. Too much caffeine or alcohol and certain drugs can also be the cause.
The heart beats when electrical signals move through it. Ventricular fibrillation is a condition in which the heart's electrical activity becomes disordered. When this happens, the heart's lower (pumping) chambers contract in a rapid, unsynchronized way. (The ventricles "flutter" rather than beat.) The heart pumps little or no blood therefore the probability of death is high.
There were a lot of patients that came into the emergency room on Friday November 20, 2015 that had heart problems. The first patient I observed was only thirty and she had been having tachycardia since two o’clock Thursday afternoon. She stated that she has had this problem before but it never lasted this long. She was said to have SVT prior to arriving to the emergency room by the ambulance crew. Upon arriving in the ER her heartrate got up to 220 beats per minutes. The next patient that I saw was brought in because his wife said that he was shaking more than usual, he had Parkinson’s. There was a young lady that came in who had hit her head almost three weeks ago and was diagnosed with a concussion at that time. She was now having neck and head pain and numbness in the back of her head. There was another assumed SVT patient. Her heartrate was around 170. There was also a homeless man that was brought in complaining of pain all over and all he kept asking for was morphine