Today I will be explaining the importance and details of tracheobronchitis also referred to as bronchitis. Tracheobronchitis as the name gives off is an inflammation of the trachea and bronchitis. The trachea and bronchi’s main role is to extend air into the lungs, so that they are able to reach the alveoli which are responsible for gas exchange in the lungs. Tracheobronchitis is often times not contagious depending upon the cause of inflammation, inflammation can result from an allergic reaction, bacterial infection or virus. Some important clinical manifestations that you may see include wheezing which are a result of inflamed airways,fever, dry or phlegm cough, night sweats, headache and sore throat. Tracheobronchitis does not always have to be severe it can also be acute and last only a few weeks. Tracheobronchitis is often times found in intubated patients who need assistance breathing by a ventilator. These infections are often times caused by multidrug resistant bacteria (methicillin resistant staphylococcus aures or gram negative bacilli), where they collect in the oropharynx and enter the respiratory by the endotracheal tube cuff or through the lumen (Craven & Hjalmarson, 2010). Ventilated associated tracheobronchitis (VAT) leads to ventilator associated pneumonia if not affectively treated with the appropriate medications and prevention techniques over time. One must understand the significance in order to properly put into effect the prevention and therapy. The endotracheal tube cuff and intralumenal biofilm formation also prevent the exit of bacteria and secretions from the lower airway, increasing the need for manual tracheobronchial suctioning (Craven & Hjalmarson, 2010). The numbers and virulence of types of pathoge... ... middle of paper ... ... bedside, and reminders to take antibiotics. It is critical in plan of discharge that the patient finishes out the antibiotic regimen if prescribed, also encourage and explain the necessity of the antibiotics in treating the condition. Lastly the patient must avoid overexertion to prevent relapse or exacerbation of the infection. Reference Craven , D., & Hjalmarson, K. (2010). Ventilator-associated tracheobronchitis and pneumonia: thinking outside the box. Clinical Infectious Diseases: An Official Publication Of The Infectious Diseases Society Of America , 1, p.S59-66. Retrieved from http://ehis.ebscohost.com/eds/detail?sid=44b983f2-9b91-407c-a053-fd8507d9a657@sessionmgr4002&vid=9&hid=116&bdata=JnNpdGU9ZWRzLWxpdmUmc2NvcGU9c2l0ZQ== Hinkle, J., Cheever, K., & , (2012). Textbook of medical-surgical nursing. (13 ed., pp. 586-588). Philadelphia: Wolters Kluwer Health
Whooping cough is a highly contagious and acute respiratory disease caused by an aerobic Gram negative encapsulated coco-bacillus bacterium, Bordetella pertussis. It is a strict human pathogen with no known animal or environmental reservoirs and an air-borne disease. On inhalation, Bordetella pertussis colonizes the ciliated cells of the bronchio-epithelium to cause disease characterised by; epithelial damage, hyper mucus secretion, pulmonary edema and paroxysmal coughing. It is often accompanied by pneumonia, otitis edema, seizures, post-tussive vomiting and encephalopathy (1).
With additional bodily fluid and less cilia to dispose it, patients build up an endless hack that raises sputum, a blend of bodily fluid and cell debris. Stagnant bodily fluid in the respiratory tract gives a development medium to microorganisms. This can prompt disease and bronchial aggravation, with side effects that incorporate dyspnea, hypoxia, cyanosis, slight fever, chills, and assaults of hacking (Saladin, 2010). In addition, bronchitis can cause shortness of breath, wheezing, chest pain, and tickle of the
Haas, D. F. (1990). The Chronic Bronchitis And EMPHYSEMA. New York,NY: John Wiley and Sons, Inc.
American Association of Nurse Anesthetists. Professional Aspects of Nurse Anesthesia Practice. Philadelphia: F. A. Davis Company, 1994. Print.
Ignatavicius, D. D., & Workman, M. L. (2013). Care of Intraoperative Patients. Medical-surgical nursing: patient-centered collaborative care (7th ed.). St. Louis: Elsevier.
Cystic Fibrosis is an inherited disease characterized by the buildup of thick, sticky mucous that can cause severe damage to the body’s organs. Mucous is usually a slippery substance that lubricates and protects the linings of the airway, digestive system, reproductive system and other organs and tissue. Problems with digestion can lead to diarrhea, malnutrition, poor growth, and weight-loss. Due to the abnormally thick mucous it can can clog airways, leading to breathing problems and bacterial infections in the lungs. Bacterial infections can lead to coughing, wheezing and inflammation. Overtime these infections can lead to permanent damage in the lungs including the formation of scar tissue, known as fibrosis and cysts in the lungs (Genetics Home Reference, 2013). The symptoms and signs of this disease vary but mostly include progressive damage to the respiratory system and chronic digestive system problems. An individuals’ lungs who are infected by cystic fibrosis have bacteria from an early stage. This bacteria can spread to the small airways, leading to the formation of bacterial micro-environments known as biofilms. Biofilms are difficult for antibodies to penetrate, therefore the bacteria repeatedly damage the lung and gradually remodel the airways, resulting in difficultly to eradicate the infection (Welsh, 1995). Cystic fibrosis patients may even have their airways chronically colonized be filamentous fungi and/or yeasts. Most men with cystic fibrosis have congenital bilateral absence of the vas deferens (CBAVD), a condition in which the tubes that carry sperm are blocked by mucous and do not develop properly. As well, women may experience complications in pregnancy. Either the c...
Person, A. & Mintz, M., (2006), Anatomy and Physiology of the Respiratory Tract, Disorders of the Respiratory Tract, pp. 11-17, New Jersey: Human Press Inc.
Liam is a previously healthy boy who has experienced rhinorrhoea, intermittent cough, and poor feeding for the past four days. His positive result of nasopharyngeal aspirate for Respiratory Syncytial Virus (RSV) indicates that Liam has acute bronchiolitis which is a viral infection (Glasper & Richardson, 2010). “Bronchiolitis is the commonest reason for admission to hospital in the first 6 months of life. It describes a clinical syndrome of cough tachypnoea, feeding difficulties and inspiratory crackles on chest auscultation” (Fitzgerald, 2011, p.160). Bronchiolitis can cause respiratory distress and desaturation (91% in the room air) to Liam due to airway blockage; therefore the infant appears to have nasal flaring, intercostal and subcostal retractions, and tachypnoea (54 breathes/min) during breathing (Glasper & Richardson, 2010). Tachycardia (152 beats/min) could occur due to hypoxemia and compensatory mechanism for low blood pressure (74/46mmHg) (Fitzgerald, 2011; Glasper & Richardson, 2010). Moreover, Liam has fever and conjunctiva injection which could be a result of infection, as evidenced by high temperature (38.6°C) and bilateral tympanic membra...
VAP develops in a patient after 48 hours or more of endotracheal intubation. According to a study by Relio et al. (as citied in Fields, L.B., 2008, Journal of Neuroscience Nursing, 40(5), 291-8) VAP adds an additional cost of $29,000-$40,000 per patient and increases the morality rate by 40-80%. Mechanically ventilated patients are at an increased risk in developing VAP due to factors such as circumvention of body’s own natural defense mechanisms in the upper respiratory tract (the filtering and protective properties of nasal mucosa and cilia), dry open mouth, and aspiration of oral secretions, altered consciousness, immobility, and possible immunosuppression. Furthermore, the accumulation of plaque in the oral cavity creates a biofilm that allows the patient’s mouth to become colonized with bacteria.
Myobaterium tuberculosis is a nonmoving, slow-growing, acid-fast rod transmitted via aerosolization (airborne route). People who are most often infected are those having repeated close contact with an infectious person who has not yet been diagnosed with TB (Ignatavicius and Workman, 2006). Therefore, when a person with active TB coughs, sneeze, laughs, sings, or whistles, droplet become air borne and may be inhaled by others. Far more people are infected with the bacillus than actually develop active TB. The bacillus multiplies freely when it reaches a susceptible site (bronchi or alveoli). The majority of the bacilli are trapped in the upper parts of the airways where the mucus-secreting goblet cells exist. An exudative response occurs, causing a nonspecific pneumonitis (Ignatavicius and Workman, 2006). With the development of acquired immunity, further growth of bacilli is controlled in most initial lesions. Bacilli can also spread by erosion of the caseating lesions into the lung airways -and the host becomes infectious to others. Cell-mediated immunity develops 2 to 10 weeks after infection and is manifested by a positive reaction to a tuberculin test. Skin testing for tuberculosis is useful test to detect. According to Universi...
Bronchiectasis is a chronic disease of the lungs where the airways of the lungs become abnormally widened, leading to a build-up of excess mucus that can make the lungs more vulnerable to infection (Nhs.uk.2014). The extent of the disease can vary greatly- may be one section of an airway that is widened and abnormal or many airways- usually somewhere between these extremes. Widened parts of the airways are damaged and inflamed, which causes excess mucus to form which is less easily cleared. Furthermore, these parts of the airways are weaker and more liable to collapse inwards which may affect airflow through the affected airways- severity depends on how many of the airways are affected (Patient.co.uk. 2014). Patients with bronchiectasis have chronic cough and excess sputum production, and infections due to bacteria develop in them- this results in the loss of lung function (O’Donnell 2008).
Pneumonia can also become a hospital acquired infection. Ventilator-associated pneumonia is a type of lung infection that occurs in a person who has been on a ventilator.... ... middle of paper ... ...
Although students were not allowed in the recovery unit, I was able to talk to one of the recovery nurses. I learned that a nurse’s duty of care includes monitoring the patient’s vital signs and level of consciousness, and maintaining airway patency. Assessing pain and the effectiveness of pain management is also necessary. Once patients are transferred to the surgical ward, the goal is to assist in the recovery process, as well as providing referral details and education on care required when the patient returns home (Hamlin, 2010).
...o Pneumonia, it causes respiratory failure. The treatment for this would most likely be ventilator breathing for the patient with supplemental oxygen. (Boothby, L. A. (2004)
...as better ventilation, negative pressure rooms and the use of UV light for disinfection, nosocomial transmissions continue to post a problem possibly because they are iatrogenic and cases may be missed due to underdiagnosis. 16