Biomedical Implants Research Paper

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Current birth control implants, antimicrobial nanoparticle coatings, and future of biodegradable implants
Clare Schmedes
First Year Seminar: Biomedical Implants
December 9th, 2014

Abstract
Birth control implants are becoming an increasingly popular method of birth control. They are preferred by some women over other contraceptive methods due to their extreme effectiveness, discreetness, fast reversibility, ability to be used while breastfeeding, and lack of medication to keep up with everyday. The implants first appeared in the 1960s and have since been improved upon to become smaller with less rods and longer lasting effectiveness. The newest and most popular models of the implant consist of an ethylene vinyl acetate copolymer plastic …show more content…

The etonogestrel is a synthetic hormone that causes changes in the users cervical mucus and uterine lining, which makes it difficult for the sperm to reach the uterus as well as inhibiting the fertilized egg to attach to the uterus. Since the hormone is distributed intravenously, it is 100% bioavailable. Etonogestrel is metabolized by the liver through the enzyme CYP3A4 and has a half-life of about 25 hours. The body needs approximately 25-30 μg/day of etonogestrel to suppress ovulation. Figure 3 shows the release pattern of etonogestrel at hours and months after insertion and removal. The peak serum concentration is shown to occur a few weeks after insertion and Implanon release 35-45μg per day at the end of the first year after insertion. Following the end of the second year that range drops slightly, releasing about 30-40 μg per day and by the end of the third year the range declines to 25-30 μg per day, which is still within the range to suppress ovulation. This shows why the implant is recommended to prevent pregnancy for three years, but needs replacement after that time. Nexplanon follows the same release pattern and timeline. After removal, the amount of etonogestrel falls rapidly and is undetectable within one …show more content…

When bacterial adhesion comes before tissue integration the host defenses are not able to prevent further colonization due to the subsequent formation of a biofilm. This leaves a window of about 6 hours from implantation that determines whether or not the implant will successfully avoid bacterial adhesion. During this window of time the implant is especially vulnerable to surface colonization. If the surface colonizes than the biofilm that follows the bacteria will be very resistant to immune response and antibiotic treatments. The biofilm is typically what causes infection due to their toughness and resistance. However, a biofilm is only able to form if bacteria have adhered so preventing bacterial adhesion could in turn prevent the biofilm from forming

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