The following questions relates to the patient within the first 24 hours 1. Outline the causes, incidence and risk factors of the identified disease and how it can impact on the patient and family (450 words) This case study is about Abdul Chidiac, a 51 year old male, married with 4 children. He had a medical history of hypertension, hypercholesterolaemia and cirrhosis with two admissions in the last six months. He is a smoker and drinks beer, 5-6 bottles per day. As Carithers & McClain (2010) explained
bleeding are hematemesis, coffee ground vomiting, melena, hematochezia (maroon coloured stool) if the hemorrhage is severe, severe syncope, chest pain, shortness of breath and anemia. Esophageal varices, esophagitis, Mallory-Weiss tears, esophageal ulcers or even esophageal cancers may cause esophageal bleeding. Esophageal varices are defined as extremely dilated and tortuous sub-mucosal veins in the lower third of the esophagus. They are most often a consequence of portal hypertension, commonly secondary
cost the healthcare system a lot of money. There is no cure for Achalasia, therefore this is something that needs to be treated, sometimes as often as every 3 months (Dugdale, George & Zieve, 2010). When my mother has the surgery to stretch her esophageal sphincter, the average cost of her surgery ranges from $1,900 to $3,000, depending on anesthetics and whether or not they need to suck out food that is stuck in the esophagus. Insurance pays the majority of the cost (Brown, 2013). Achalasia effects
six-packs of beer and a pint of vodka on a regular basis for years. This is the second time he has presented with this symptom. Last week, he was admitted in to the same hospital for the same complaint. An endoscopy was performed, which revealed esophageal varices. Banding was done and he was discharged yesterday with no post-operative complications. He suddenly began vomiting blood today in the afternoon. He denies any trauma, eating, or taking any medication prior to the current episode. His physical
Wilson disease What is Wilson disease? Wilson disease is a medical condition characterized by liver, neurologic, and psychological abnormalities related to the deposition of copper in the liver and brain. The condition occurs due to impaired copper metabolism, which result in copper build up in tissues. Most patients are diagnosed at age 5-35. Untreated Wilson disease frequently results in liver injury that often advances to liver cirrhosis and end-stage liver disease. Copper accumulation in
DEFINITION (1) Cirrhosis can be simply defined as diffuse fibrosis associated with parenchymal nodules. It involves the whole liver rather than a small part even though on occasion the fibrosis may be more severe in one or other lobe. Fibrosis results partially from collapse of the new fibre formation and is usually regarded as a consequence of liver-cell necrosis. The nodules of a cirrhotic liver are the surviving parenchyma. But they are strutually altered. They vary greatly in size and appearance
The Author known as, Scott Fitzgerald is considered a notable writer in the Jazz Age time period for his novels, “short stories” such as “The Great Gatsby.” Throughout the life of Scott Fitzgerald his works have proven to be of high merit that have impacted the world of literature In 1898 at Saint Paul, Minnesota, born into a middle-upper class family, Fitzgerald was named after his famous second cousin Francis Scott Key but he finally went by the familiar name Scott Fitzgerald. Also he was named
ince thiamine is an important cofactor in the pyruvate dehydrogenase complex, a deficiency caused by excessive alcohol intake can have devastating effects on the nervous and cardiovascular systems. Neurological effects caused by thiamine deficiency are known as Dry beriberi, while cardiovascular effects are known as Wet beriberi (Morse 1992). Thiamine deficiency can result in acetylcholine deficiency, which leads to memory loss and lack of concentration. Thiamine is responsible for the maintenance