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Rosacea peculiar dermatit
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Rosacea is another chronic skin disorder whose symptoms include abnormal vascular and inflammatory conditions. Clinical manifestations might be severe and include flushing, facial erythema, inflammatory papules and pustules, telangiectasias, edema, and watery or irritated eyes. (DOI: 10.4155/cli.11.50,Issn Print: 2041-6792). Taking into account many factors, four main classes of rosacea were distinguished: erythematotelangiectatic, papulopustular, phymatous, and ocular . (PMID: 14959940)
Patients with the first subtype suffer from flushing and persistent facial erythema, sometimes accompanied by telangiectasias. The second subtype of rosacea is characterized by the presence of papules and pustules. Patients with the phymatous subtype show symptoms
Arch Dermatol. 2007;143(1):124–125. Puchenkova, S. G. (1996). "
Barone, Eugene J., Judson C. Jones, and Joann E. Schaefer. "Hidradenitis Suppurativa." Skin Disorders. Philadelphia: Lippincott Williams & Wilkins, 2000. 21-25. Print.
that cause dark red blotches on the skin, usually on the face). Laboratory tests are another consideration of diagnosis. Tests consist of anti-nuclear anti-body (ANA) counts and anti-topoisomerase (an enzyme that reduces super-coiling in DNA by breaking and rejoining one or both strands of DNA). High ANA’s and low anti-topoisomerases are found in patients with Raynaud syndrome. (Desai, 2003) “Patients with circulating autoantibodies, antinuclear antibodies, and anti-Scl 70 antibodies are at (an) increased risk of developing a connective tissue disease. Systemic sclerosis is the connective tissue disease most frequently associated with Raynaud’s phenomenon.” (Bowling, 2003) This syndrome is described as primary Raynaud phenomenon (PRP) if is not associated with another disorder and as secondary Raynaud phenomenon (SRP) if it occurs in association with another disorder.
...may have the same symptoms. The symptoms are red bumps that may bleed if the sores are picked over.
HZ patients typically present with a characteristic, distinguishing unilateral, localized, vesicular eruption in dermatome distribution, that is often followed by an intensive localized prodromal pain. In rare instances, it is acco...
In the 1990s, however, Bastuji and Roujeau each proposed that erythema multiform major and Stevens-Johnson syndrome are 2 distinct disorders (Roujeau, 1997). They suggested that the diagnosis of erythema ...
The history of urticaria is a fascinating account of mankind gradually growing to understand human diseases. The process starts with the many different names this disease has been given to in the past. The school of Hippocrates in 4th century BC recognized urticaria with nettles. The roman school instead focused on its burning sensation and is where Plinius named it uredo. In the 10th century, Hali Ben Abbas used the name essera because it means mountain or elevation, alluding that the welt is above skin level. In 18th century, Zelder called it urticatio, and in 1792, urticaria was the accepted name. A couple of words have been made during recent times but they did not clearly distinguish the disease from the other skin diseases. Some writings of the 16th century can be rediscovered indicating that strawberries and shellfish were implicated as a cause for urticaria. There were many other theories that tried to explain the causes of the formation of these raised welts. Many stated that certain foods were the reason but slowly doctors discovered that it is not just food that triggers urticaria.
Schepis, Carmelo, Donatella Greco, and Corrado Romano. "Cardiofaciocutaneous (CFC) Syndrome." Australasian Journal of Dermatology 40.2 (1999): 111-13. Print.
Epidermolysis Bullosa also known as E.B. is generally an inherited connective tissue disease. This disease is evident at birth or soon there after. It causes large fluid filled blisters, in the skin and mucosal membranes. Chaffing or even increase in room temperature may cause these blisters to form. E.B. affects an estimated 50 in 1 million live births. The disease has been known to affect every racial and ethnic group and is found in both males and females all over the world. The disease has been seen in a wide variety of forms from mild to lethal form involving some organs. Epidermolysis Bullosa is the result of a mutation in the keratin or collagen gene. There are three layers of skin epidermis, dermis and the subcutaneous layers. Epidermolysis Bullosa affects the top two layers. For those who are not affected by E.B. there are protein anchors that prevent the top two layers from moving without unison or shearing. But those who are affected by E.B. lack the protein anchors that are made of collagen. This is what creates the friction between the layers and this rubbing and pressure causes the painful blisters and sores which have been compared to third degree burns. There are four main types of Epidermolysis Bullosa: Dystrophic Epidermolysis Bullosa, Epidermolysis Bullosa simplex, Hemindesmosomal Epidermolysis Bullosa and Junctional Epidermolysis Bullosa. Even within the main types there are many sub types including Epidermis Bullosa Acquisita generally appearing in adults over the age of 50. This specific type is also linked to Cohen’s disease and Lupus.
This condition can affect the face (called keratosis pilaris atrophicans faciei), neck (called erythromelanosis follicularis faciei et colli), eyebrows (called Ulerythema ophryogenes), scalp (called keratosis follicularis spinulosa decalvans), and cheeks (called atrophoderma vermiculata / folliculitis ulerythematosa reticulata or pitted scarring of cheeks).
There are 5 main types of psoriasis and they are: plaque, guttate, inverse, pustular, and erythrodermic. The type of psoriasis is diagnosed based on the appearance and can be confirmed with either skin biopsy or the patient’s medical history. Skin biopsy involves the removal of the patient’s skin sample to be analyzed under a microscope and base on the appearance to determine the type of skin disease the patient has. Psoriasis tends to occur within families that have it. Plaque psoriasis or psoriasis vulgaris is the most common type of psoriasis and is responsible for 80% of all patients with psoriasis. Young and middle aged adults are most affected by this type of psoriasis. People with plaque psoriasis experience raised red scaly lesions on their body that tend to itch and crack to bleed. These patches are generally known to appear on the knees, elbows, scalp, and the back but they may appear anywhere on the skin. Around 10 to 20 percent of patients with plaque psoriasis will develop psoriatic arthritis in their life time. Guttate psoriasis usually target people younger than 30 years...
Liotta, MD, Elizabeth A., and Dirk M. Elston, MD. "Dermatologic Aspects of Addison Disease." MedScape. WebMD, 8 Mar. 2013. Web. 25 Jan. 2014.
The symptoms of psoriasis differ from type to type, although inflamed, scaly lesions are present in all five types. The most common form of the disease, plaque psoriasis, is identified by small bumps that begin to grow and become scaly. These lesions flake easily, but removing these patches can cause the tender skin below to bleed. In the Guttate type, small, individual, red drops form. This type does not have as much scaling as plaque psoriasis. The drops usually clear up on their own, but may also reappear as a different form of psoriasis, usually plaque. Inverse psoriasis usually occurs in places where the skin folds, such as the genitals, breasts, armpits or the backs of knees. This type will appear red, yet it will be smooth and dry. Also, no scaling will occur. Pustular psoriasis is a type that's significantly more rare. It is also more painful. In this type, blisters filled with non-infectious pus appear within a few hours and then dry up and peel within another two days. Severe medical risks exist for those who have this particular form of psoriasis, due to its side effects; exhaustion, anemia, weight loss, fever, chills, rapid pulse rate, severe itching and muscle weakness. Even less common than pustular psoriasis is erythrodermic psoriasis. This type is...
The condition may be present at birth or in the very early years of the child’s life. Besides the obvious polygon shaped scaly patches on the skin(gray, white, brown) other notable and identifiable symptoms include itchy, dry, or thick skin. However, Mr.Anders and Mrs.Anders, I am not trying to send you into a panicked alarm as there is much to be done for the child in the future. Ichthyosis Vulgaris does not affect life expectancy in any manner and although there is no cure for the condition certain treatment can help manage the
Systematic lupus erythematosus (SLE) is a chronic, multi-organ, auto-immune, inflammatory disease that is marked by exacerbation and remission periods. The term is derived from the characteristic of the malar (butterfly) rash, which is present in individuals with SLE. Only 8-15% of children are affected, so it predominantly occurs in adults, customarily in women of the childbearing age (20-40 years), with ratios of 9:1 for females and 15:1 for males. African-American women are three to four times more likely to develop SLE than Caucasian women, and those of Hispanic,Native American, Asian, and Afro-Caribbean decent have a higher incidence rate than those individuals of Caucasian race. This only applies for SLE, because those who encounter drug-induced lupus erythematosus (DILE) are more frequently Caucasian than Afri...