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Neurological diseases quizlet
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Neurological degenerative diseases such as dementia have detrimental effects on the lives of people that suffer from them such as memory and concentration loss. These diseases are usually caused by genetic disposition, trauma, or stroke, though symptoms similar to those in dementia patients may also be a result of synaptic quantum tunneling of neurotransmitters such as glutamate and norepinephrine which are responsible for memory and concentration respectively.
During the process of neuron excitation and firing, neurotransmitters are released by the axon terminal of one neuron in order to excite adjacent neurons. When memories are being recalled, higher amounts of the neurotransmitter glutamate are released between neurons involved in that
When a chemical signal is transmitted, the presynaptic neuron releases a neurotransmitter into the synapse. The signal is then sent to the postsynaptic neuron. Once the postsynaptic neuron has received the signal, additional neurotransmitter left in the synapse will be reabsorbed by the presynaptic
Dementia is a long-term condition that normally affects people aged 65 and over, younger people can be affected. Having dementia can cause loss of key functions to the brain, such as; loss of memory; confusion; speech and language problems; loss of ability to make judgements; loss of concentration; difficulty in processing information; changes in behaviour and personality. These all lead to a person not been able to function properly. The person’s ability to function deteriorates over a period of time and is usually at least 6 months before positive diagnosis of dementia can be made. Dementia is caused when the brain is damaged by diseases such as Alzheimer’s which is the most common of dementia, vascular which is a series of mini strokes,
Lewy body dementia (LBD) is a progressive dementia and the second most common type of dementia after Alzheimer's disease dementia. Protein deposits, called Lewy bodies, develop in nerve cells in the brain regions involved in motor control such as, thinking, memory and movement (Mayo Clinic). LBD has similar symptoms with both Alzheimer's disease and Parkinson's disease and is often misdiagnosed. Of particular note, Robin Williams suffered from this debilitating disease which lead to his suicide in 2014. It was not discovered until his autopsy three months after his death.
This essay will discuss the form of Alzheimer’s dementia Posterior Cortical Atrophy, from the aspects of the journey by Sir Terry Pratchett. In addition the essay will discuss symptoms of the disease, pathology, and anatomical structures and locations.
Parkinson's is an idiopathic, multifactorial neurodegenerative disease that attacks neurotransmitters in the brain called dopamine. Dopamine is concentrated in a specific area of the brain called the substantia nigra. The neurotransmitter dopamine is a chemical that regulates muscle movement and emotion. Dopamine is responsible for relaying messages between the substantia nigra and other parts of the brain to control body movement. The death of these neurotransmitters affects the central nervous system. The most common symptoms are movement related, including shaking, rigidity, slowness of movement and difficulty with posture. Behavioral problems may arise as the disease progresses. Due to the loss of dopamine, Parkinson's patients will often experience depression and some compulsive behavior. In advanced stages of the disease dementia will sometimes occur. The implications of the disease on the anatomy and physiology of the respiratory and phonatory systems significantly control speech.
Thesis/Preview Statement – Alzheimer’s disease (AD) causes a decline in brain function, it destroys healthy nerve cells. Today, we have discussed Causes, Symptoms, and Diagnosis of AD.
Dementia can occur in relation to many different illnesses. Some of the most common of which are Huntington’s Disease,
Acetylcholine is possibly the most widely used neurotransmitter in the body, and all axons that leave the central nervous system (for example, those running to skeletal muscle, or to sympathetic or parasympathetic ganglia) use acetylcholine as their neurotransmitter. Within the brain acetylcholine is the transmitter of, among other neurons, those generating the tracts that run from the septum to the HIPPOCAMPUS, and from the nucleus basalis to the CEREBRAL CORTEX -- both of whbasalis to the CEREBRAL CORTEX -- both of which seem to be needed to sustain memory and learning. It is also the neurotransmitter released by short-axon interneurons of the BASAL GANGLIA.
Parkinson disease (PD), also referred to as Parkinson’s disease and paralysis agitans, is a progressive neurodegenerative disease that is the third most common neurologic disorder of older adults. It is a debilitating disease affecting motor ability and is characterized by four cardinal symptoms: tremor rigidity, bradykinesia or kinesis (slow movement/no movement), and postural instability. Most people have primary, or idiopathic, disease. A few patients have secondary parkinsonian symptoms from conditions such as brain tumors and certain anti-psychotic drugs.
Dementia is the progressive deterioration and impairment of memory, reasoning, and other cognitive functions occurring as the result of a disease or condition. Dementia can affect the person’s ability to carry out daily activities. For example, the person may forget where they live or they might think they have already done their activities but never did. Dementia can also cause the elderly to become incontinent and can’t control their urinary system. Many people get confused that dementia is a disease. Dementia is not a disease. However, it can lead to a disease or condition. Dementia is more common in the elderly population. It’s normal for people to forget things, but to a certain extent it becomes a critical issue. Depression also plays a role in the affects of dementia. Studies have been made to believe that the biological mechanisms for depression relating to dementia is, “interactions with vascular diseases, changes in glucocorticoid steroid levels that can result in hippocampal atrophy, accumulation of amyloid-[beta] plaques, inflammatory processes, and lack of nerve growth factors” (Heser et al., 2013). Dementia is caused because of plaques and neurofibrillary tangles. This can also be known as Alzheimer’s Disease. Dementia is the leading cause for Alzheimer’s Disease in the elderly. For all dementia cases, 60 to 80 percent of people with dementia will have Alzheimer’s Disease. The disease has 3 different stages, the early stage, the middle stage, and the late stage. Each of those stages has a variety of symptoms that affects the memory impairment of the person (Wieregna, Bondi, 2011). Also relating to dementia is Parkinson’s Disease and Huntington Disease. These diseases can result in impairment, which can cause challeng...
James Parkinson first discovered Parkinson's Disease in 1817. Parkinson's Disease is a common neurologic disorder for the elderly. It is a disorder of the brain characterized by shaking and difficulty with walking, movement, and coordination. This disease is associated with damage to a part of the brain that controls muscle movement. Parkinson's Disease is a chronic illness that is still being extensively studied.
So, as we can see here synaptic loss is not just age related and we can see this relationship through the evidence of the parts of the brains affected as well as a study that looks at an actual comparison of synapses. The comparison is between people with no cognitive impairment, mild cognitive impairment, and early Alzheimer’s disease. Mild cognitive impairment is a type of impairment within the brain that can cause a slight but noticeable decline in cognitive abilities. These abilities include memory and thinking skills. These patients are at a higher risk of developing Alzheimer’s and that is why they were also looked at in this research (Scheff, Price, Schmitt & Mufson, 2005). One of the major research findings that led to this study was that patients with Alzheimer’s disease seemed to have a loss of synaptic contacts in their neocortex and hippocampus. This loss of synaptic contacts demonstrates an association with cognitive ability and correlates strongly with dementia. It was unknown whether patients with mild cognitive impairment had significant synaptic loss compared to those with no cognitive impairment.
...ical impulse, repeating the mechanism described above. The neurons received signal, they crumble up the information passed it down until they get to the last one.
Creutzfeldt-Jakob Disease is an uncommon, deteriorating, consistently fatal brain disorder that is caused by prions. The symptoms of CJD are similar of Alzheimer’s but progress much faster. There are three variations of CJD, sporadic, familial, and acquired. All variations affect the brain the same way and have the same result of death. CJD is an untreatable and incurable disease.
Paramedics are frequently presented with neurological emergencies in the pre-hospital environment. Neurological emergencies include conditions such as, strokes, head or spinal injuries. To ensure the effective management of neurological emergencies an appropriate and timely neurological assessment is essential. Several factors are associated with the effectiveness and appropriateness of neurological assessments within the pre-hospital setting. Some examples include, variable clinical presentations, difficulty undertaking investigations, and the requirement for rapid management and transportation decisions (Lima & Maranhão-Filho, 2012; Middleton et al., 2012; Minardi & Crocco, 2009; Stocchetti et al., 2004; Yanagawa & Miyawaki, 2012). Through a review of current literature, the applicability and transferability of a neurological assessment within the pre-hospital clinical environment is critiqued. Blumenfeld (2010) describes the neurological assessment as an important analytical tool that evaluates the functionality of an individual’s nervous system. Blumenfeld (2010) dissected and evaluated the neurological assessment into six functional components, mental status, cranial nerves, motor exam, reflexes, co-ordination and gait, and a sensory examination.