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Cholecystitis preoperative care
Observation paper on laparoscopic cholecystectomy
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Recommended: Cholecystitis preoperative care
PREOPERATIVE DIAGNOSIS: Acute cholecystitis. POSTOPERATIVE DIAGNOSIS: Acute cholecystitis. PROCEDURE: Laparoscopic cholecystectomy. SURGEON: Dr. ___[NAME]. DESCRIPTION OF PROCEDURE: The patient was taken to the operating room and after induction a general endotracheal anesthetic, the abdomen was prepped and draped in a sterile manner. Then, 0.5% Marcaine was injected superior to the umbilicus and a longitudinal skin incision was made and carried down to the anterior abdominal wall fascia which was incised in the midline. Two stay sutures of 0 Vicryl placed on either side of the fascial incision. The peritoneum was entered under direct vision with Mayo scissors. A Hassan trocar is inserted into the peritoneal cavity and secured to the …show more content…
fascia, which was previously placed stay sutures. The peritoneum was inflated to a pressure of 15 mmHg with carbon dioxide.
The laparoscope was inserted and the remaining ports were placed visualizing their placement with a laparoscope. An 11 mm port was placed in the subxiphoid location, and two 5 mm ports were placed laterally under the right subcostal (rib) margin. The gallbladder was identified. It was edematous, acutely inflamed. It was grasped with the fundus and retracted in a (cephalad) direction. There were no omental adhesions adherent to the gallbladder that were taken down sharply. The neck was then grasped along the lateral most ports and retracted in a lateral direction. The cone bile duct was identified and care taken to avoid injury to this structure. The cystic artery and cystic duct were identified, mobilized, doubly ligated with endoclips and then divided. The gallbladder was dissected free from the liver with the electrocautery. The specimen was placed in an (Endo Catch) (sac), and was removed through the umbilical incision with no difficulty. The Hassan trocar was reinserted into the abdomen, reinflated the suprahepatic lymphatic space was irrigated copiously with normal saline. Adequate hemostasis was obtained in the gallbladder fossa with the electrocautery. The 360 degree
view of the abdomen was made with the scope. There were some adhesions in the right lower quadrant. The stomach and duodenum appeared normal, as did transverse and sigmoid colons. Trocars were then removed, while visualizing their removal with the laparoscope. No bleeding was noted from any of the trocar sites, intraperitoneally. The Hassan trocar was removed last. The fascia at the umbilicus closed with a Vicryl figure of 8 sutures. The skin incision was closed with 4-0 Monocryl (running) subcuticular sutures. The (wounds) were (dressed) and the patient was taken to the recovery room in good condition.
The gallbladder according to Dorland’s Medical Dictionary is ‘the pear shaped reservoir for the bile on the posteroinferior surface of the liver, between the right and the left quadrate lobe, from its neck the cystic duct projects to join the common bile duct’. The function of the gallbladder in the human body is to solve and concentrate bile, which is produced by the liver and is necessary for proper digestion of fats.
Mink Intro – External Anatomy Overview. (n.d.). mreroh.com . Retrieved May 27, 2014, from http://www.mreroh.com/student/apdocs/Dissection/Intro%20-%20External%20Anatomy.pdf
- If all of the options were explored, and patient is given antibiotics and is treated without any pain or suffering than the treatment identifies with the ethnical principles of autonomy, non-maleficence, and veracity. In turn, Mrs. Dawson will be happy with the outcome of the procedure.
The first laparoscopic cholecystectomy (LC) using keyhole approach was done by Professor Mouret of Lyon, France in 1987, when he was completing a gynecologic laparoscopy on a woman also suffering from symptomatic gall stones, he removed it laparoscopically instead of opening up. Dr. Eddie Reddick reported 100 cases of laparoscopic cholecystectomy in 1989. The classical four port technique of LC as described by Reddick became the most widely adopted technique.
1. Outline the causes, incidence and risk factors of the identified disease and how it can impact on the patient and family (450 words)
Cholesteatoma is a growth of excess skin or a skin cyst (epithelial cyst) that contains desquamated keratin and grows in the middle ear and mastoid (Thio, Ahmed, & Bickerton, 2005). A cholesteatoma can grow and spread, destroying the ossicles, tympanic membrane and other parts of the ear. They appear on the pars flaccida and pars tensa sections of the tympanic membrane. A cholesteatoma can occur when a part of a perforated tympanic membrane is pushed back into the middle ear space, debris and skin cells can build up forming a growth. It can obstruct tympanic membrane movement and movement of the ossicles. As the layers grow, the amount of hearing loss can increase. A cholesteatoma can be congenital (present at birth) or be acquired as a result of another disease. They can also be formed as a result of a surgery, trauma, chronic ear infection, chronic otitis media, or tympanic membrane perforation. It can develop beyond the tympanic membrane and cause intracranial and extracranial complications. Due to this patients can experience permanent hearing loss as a result of an infection of the inner ear as well as other serious health concerns. These include dizziness, facial nerve weakness and infections of the skull (Hall, 2013). Patients may present chronically discharging ear, hearing loss, dizziness, otalgia (ear pain), and perforations (marginal or attic).
One or more gallstones erode into the gastrointestinal tract, creating a cholecystenteric fistula, most commonly between the gallbladder and the duodenum. Gallstones less than 2 to 2.5 cm generally pass into the intestine without causing obstruction while stones 5 cm or larger are more likely to impact usually at the distal ileum, the narrowest part of the small bowel. Other reported sites of impaction include proximal ileum, jejunem, colon, and rarely the duodenum or stomach (bouveret’s syndrome). [11] In our case, a large, approximately 5 cm, gallstone was found impacted at the jejunum while a smaller stone was found impacted at a Meckel’s
Dr. Nolen's purpose for writing "The First Appendectomy," which speaks about his first execution of an appendectomy, was to inform. Dr. Nolen writes explains the procedures in performing an appendectomy and the possible complications that can and did occur during his first operation. Dr. Nolen informs readers that, “There are five layers of tissue the abdominal wall: skin, fat, fascia (a tough membranous tissue), muscle
At 2:44pm Arlethea from Cardinal innovations contacted Mobile Crisis Management (MCM) in regards to requesting services for Mr. Shane Edmonds. Arlethea reported Mr. Edmonds is requesting assistance with his opiate abuse. Dispatcher contacted Qualified Professional (QP) to respond to 4615 Siler City Snow Camp Rd. Siler City, NC 27344. QP spoke with Bill Cook from Carinal Innovations to schedule estimated time of arrival of 4:00pm, which QP arrived at 4:10pm due to traffic. QP contacted Cardinal innovations upon arrival and MCM dispatcher.
On my first clinical rotation outside of 5w, in the Roanoke Memorial Hospital, I had the pleasure of visiting the OR. My last week of clinical rotation, I got the opportunity to witness two different cases. I saw a hemorrhoidectomy, and a Laparoscopic colectomy. Although I only had an opportunity of witnessing the hemorrhoidectomy in the middle of the procedure, both procedures were quite invasive. There were both very interesting to watch.
What is hepatic cirrhosis? According to the medical dictionary hepatic cirrhosis is when scar tissue replaces the liver’s healthy tissue. This disease changes the structure of the liver and blood vessels that nurture it. It reduces the liver’s capability to produce proteins and process hormones, nutrients, medications, and poisons. Cirrhosis is an illness that gets worse over time and possibly can become life threatening. This serious illness is ranked as the ninth leading cause of death in the U.S. It is the third most common cause of death for adults between 45 and 65 years of age. It occurs in more than fifty percent of undernourished chronic alcoholics. Unfortunately it kills about 35,000 people per year. In other country’s such as Africa and Asia death from cirrhosis is usually caused by hepatitis B.
Sephton (2009) discusses an overview of severe ulcerative colitis, along with assessments, medical management, and nursing care. Ulcerative colitis is an inflammation in the mucosal layer of the colon. Ulcerative colitis has characteristics of watery diarrhea with blood, mucus, or pus. Treatment usually depends on the extent and severity of the disease. Mild to moderate ulcerative colitis is treated with 5-aminosalicylic acid. Corticosteroids can be used for patients who relapse or do not respond well to the 5-aminosalicylic acid treatment. Azathioprine or 6-mercaptopurine are immunosuppressive drugs that are used when the disease becomes steroid dependent. For patients with severe ulcerative colitis, intravenous corticosteroids during a hospital stay are used.
The pancreas is located in the middle of the abdomen. It’s surrounded by the stomach, small intestine, liver and spleen. It’s about six inches long and shaped like a thin pear, wide at one end. It has three sections: wider right end is the head, the middle is the body and the left end is the tail.
Ulcerative Colitis is a chronic inflammatory bowel disease (IBD) of the large intestine (colon). Ulcerative colitis only affects the colon, it causes the colon to become inflamed and develop ulcers along the lining of the large intestine. Patients can develop colorectal cancer (bowel cancer) from having extensive ulcerative colitis. Cells and proteins in a healthy immune system protect people from the infection. Patients suffering from ulcerative colitis dysplasia have an abnormal immune system. The body may mistake food or other materials as invading/foreign substances and will send in white blood cells into the lining of the intestines (colon). Ulcerations along with chronic inflammation are serious symptoms
The patient has experienced fever, chills on body, headaches and anorexia as well as sweating especially during the night. The patient has also been feeling fatigued, muscle aches and nausea as well as vomiting especially after eating (WHO, 2010, p. 117). These symptoms started forty eight hours ago, and the patient has not taken any medication except for some aspirin.