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Essay on cardiopulmonary resuscitation
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Essay on cardiopulmonary resuscitation
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1. What are the compensatory mechanisms that occur when a patient has heart failure?
Sympathetic nervous stimulation: This compensatory mechanism is the first one. Epinephrine (adrenaline) and norepinephrine (noradrenaline) are released which causes an increase in BP and the heart to pump faster and more powerfully. They also increase cardiac output in this way it temporally helps compensate for the hearts reduced ability to pump blood. This is not good long term as it increased the amount of oxygen the myocardium needs.
Renin angiotensin system activation: Because of decreased blood flow to the kidneys the compensatory mechanisms activate to hold on to sodium and water. When the Blood flow is decreased Angiotensin II is released causing vasoconstriction
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This also caused an increase in demand of oxygen to the cardiac muscle so it’s not a good thing (Ignatavicius &Workman, 2013, p. 747).
2. What are 3 things that can worsen heart failure and why?
High sodium diet: Too much sodium causes fluid retention which increases the workload of the heart. The reason sodium causes water retention is because water moves from low concentrations to high concentrations. Water follows salt so when too much salt is consumed it draws the water from the blood into the tissue.
Not taking the prescribed medication: Medications are prescribed to decrease the workload of the heart, prevent fluid retention and decrease the amount of harmful hormones that are released. When the patient fails to comply with prescribed medications all these things are not managed.
Not staying active: Because it has been proven that activity can help with weight loss, lower blood pressure, reduce LDL cholesterol and increase HDL cholesterol, all of these can decrease the workload of the
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It occurs because of repetitive electrical activity. This can occur in a patient with early or late heart failure, because there is damage to the heart tissue and the heart beats faster to try to supply the body with blood. Recommended treatment is elective cardioversion. Drugs used include an antidysrhythmic such as Mexitil or Sotalol (Ignatavicius &Workman, p. 728-729).
4. What are the primary assessments for a patient in heart failure?
A cardiac assessment: Listen to heart sounds listening for extra heart sounds, fast heartbeat, and monitor EKG looking for dysthymias. Assess vitals especially BP, BP should be kept low in heart failure patients to put less stress on the heart. Assess the patient for edema as a result of fluid retention. Listen for crackles in the lungs due to fluid built up. Watch I&O’s and weight the patient to assess for edema, ask about activity intolerance. Assess for changes in mental status, cool extremities, pale or cyanotic, fatigue, and JVD (Indications of poor perfusion) (Ignatavicius &Workman, p.756).
5. What is the primary nursing diagnosis for this
The experiment studies the effects of Red Bull and its major components on the heart rate of a Daphnia. The experiment focuses on the effects of conditions on the cardiovascular system. The Cardiovascular system is responsible for the transport of blood, oxygen, nutrients and waste circulating the body. It consists of the heart, vessels, and blood as in closed circulatory system and hemolymph in open circulatory system, the cardiovascular system is also responsible for thermoregulation in the body. (Gonzalez, 2012). The heart helps pump blood to the lungs and rest of the body. The pumping of heart or the contraction and relaxation of heart determines the heart rate and depends on multiple chemicals that we could influence by using stimulants, depressants, varying temperatures, aerobic, and anaerobic
When a muscle contracts and relaxes without receiving signals from nerves it is known as myogenic. In the human body, the cardiac muscle is myogenic as this configuration of contractions controls the heartbeat. Within the wall of the right atrium is the sino-atrial node (SAN), which is where the process of the heartbeat begins. It directs consistent waves of electrical activity to the atrial walls, instigating the right and the left atria to contract at the same time. During this stage, the non conducting collagen tissue within the heart prevents the waves of electrical activity from being passed directly from the atria to the ventricles because if this were to happen, it would cause a backflow. Due to this barrier, The waves of electrical energy are directed from the SAN to the atrioventricular node (AVN) which is responsible for transferring the energy to the purkyne fibres in the right and left ventricle walls. Following this, there is a pause before the wave is passed on in order to assure the atria has emptied. After this delay, the walls of the right and left ventricles contract
This systematic review conducted by Takeda A, Taylor SJC, Taylor RS, Khan F, Krum H, Underwood M, (2012) sourced twenty-five trials, and the overall number of people of the collective trials included was 5,942. Interventions were classified and assessed using the following headings.-
...dwin M, Parlow JL. Effects of low-intensity exercise conditioning on blood pressure, heart rate, and autonomic modulation of heart rate in men and women with hypertension. 2009 Oct; Vol. 11 (2), pp. 129-43. Date of Electronic Publication: 2009 Jan 15. Ebscohost. Available from: http://web.b.ebscohost.com.proxy.elmhurst.edu/ehost/detail?sid=e45c21d6-7074-4dc5-8390-f4e832d5c470%40sessionmgr110&vid=1&hid=126&bdata=JnNpdGU9ZWhvc3QtbGl2ZQ%3d%3d#db=mnh&AN=19150992
... uptake during submaximal exercise but did increase heart rate and the rate-pressure product at rest and during both exercise and recovery’.
An electrocardiogram (ECG) is one of the primary assessments concluded on patients who are believed to be suffering from cardiac complications. It involves a series of leads attached to the patient which measure the electrical activity of the heart and can be used to detect abnormalities in the heart function. The ECG is virtually always permanently abnormal after an acute myocardial infarction (Julian, Cowan & Mclenachan, 2005). Julies ECG showed an ST segment elevation which is the earliest indication that a myocardial infarction had in fact taken place. The Resuscitation Council (2006) recommends that clinical staff use a systematic approach when assessing and treating an acutely ill patient. Therefore the ABCDE framework would be used to assess Julie. This stands for airways, breathing, circulation, disability and elimination. On admission to A&E staff introduced themselves to Julie and asked her a series of questions about what had happened to which she responded. As she was able to communicate effectively this indicates that her airways are patent. Julie looked extremely pale and short of breath and frequently complained about a feeling of heaviness which radiated from her chest to her left arm. The nurses sat Julie in an upright in order to assess her breathing. The rate of respiration will vary with age and gender. For a healthy adult, respiratory rate of 12-18 breaths per minute is considered to be normal (Blows, 2001). High rates, and especially increasing rates, are markers of illness and a warning that the patient may suddenly deteriorate. Julie’s respiratory rates were recorded to be 21 breaths per minute and regular which can be described as tachypnoea. Julies chest wall appeared to expand equally and symmetrical on each side with each breath taken. Julies SP02 levels which are an estimation of oxygen
This only keeps the blood moving for a short while. The heart muscle walls weaken and are unable to pump as strongly. This makes the kidneys respond by causing the body to retain fluid and sodium. When the body builds up fluids, it becomes congested. Many conditions can cause heart failure, and they include coronary artery disease, heart attack, cardiomyopathy, and conditions that overwork the heart.
Heart failure is a major clinical, social and economic problem in the United Kingdom according to the Department of Health [DH] (2013).The National Institute for Health and Clinical Excellence [NICE] (2010) reported that about 900,000 people suffer from heart failure in the United Kingdom. The National Institute for Cardiovascular Outcomes Research [NICOR] (2011) conducted a national audit which found that one in every 20 people over the age of 65 is diagnosed with heart failure which demonstrates that it mainly affects the elderly. As a leading cause of mortality, heart failure contributes to more than 6,000 deaths each year (NICOR, 2011). Newly diagnosed cases of heart failure have a 40% risk of dying within a year (NICOR, 2011). Despite advances in therapy, mortality is still high and only half of patients are alive five years after being diagnosed with heart failure (NICE, 2010).
Sympathetic pathways change nerve activity during times of stress, exercise, low blood glucose levels, excitement or fear, due to the flight or fight response. These changes can have an effect on homeostasis by increasing heart rate, increasing blood flow, dilating pupils, sweating, releasing glycogen, increasing oxygen intake and diverting blood flow away from the gastrointestinal tract.
Reduce salt intake. Too much salt impacts the ability of your kidneys to remove water from the bloodstream. The result of this is higher blood pressure because of the extra fluid. Stop using table salt and reduce the amount of salt you use when cooking.
Our heart is like a double pump mechanism where the right side pumps deoxygenated blood to our lungs and the left side pumps oxygen rich blood to circulate back through our bodies. This process provides a continuous supply of oxygen and eliminates carbon dioxide waste from our body. When there is a disturbance to this process, it causes our heart the inability to pump sufficiently to meet our body’s demands. This causes the body to accumulate blood and fluid in the organs and tissues and will lead to impaired circulation or congestive heart failure. Congestive heart failure is not only the inability to maintain the adequate oxygen delivery; it’s also systemic in attempting to compensate for inadequacy. In general, there have been many great
The kidneys, a major organ, are responsible for the control of blood pressure. When the pressure of blood flow is continually high, blood vessels can stretch, scar, weaken, or even harden (citation?). Whether blood pressure hardens or weakens the arteries and vessels is irrelevant because the end result is the same; the kidneys’ ability to filter the blood is tainted and they may stop removing sodium, wastes, and fluid from the body. The toxic substances and the fluid that is retained in the bloodstream may damage the vessels even more, leading to a dangerous downward spiral. In fact, high blood pressure is the second leading cause of kidney failure in the United States after diabetes (citation? NKUDIC). Preventing hypertension, and if present controlling it, is paramount to maintaining the health of our kidneys.
Once a sympathetic stimulation is and/or heart rate increases, stroke volume increases with more force (Frank Starling mechanism) with intensities (depending on body positions) and typically plateaus.
There are several causes of high blood pressure. One of the major causes of high blood pressure is poor diet. If there is an extensive unhealthy intake of food then that possibly means that large amounts of sodium are being ingested. The recommended daily intake of sodium is on thousand five hundred mg. meals that contain a lot of sodium also tends to have large quantities of saturated fat and cholesterol. Saturated fats and cholesterol serves to clog the arteries making the heart work extremely hard just to pump blood in the circulatory system. This causes blood pressure levels to rise (Scott).
The first compensatory mechanism is tachycardia and increased contractility that is caused by the activation of the sympathetic nervous system. This increase in heart rate and strength of contraction improves cardiac output and help maintain blood pressure. However, when the heart is beating to rapidly, blood is being pumped less efficiently. Similar because the heart is beating faster and harder, the myocardium requires more oxygen. If ischemia and hypoxia occur for a prolong period of time a myocardial infarction will