Food Addiction In Food

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Introduction The concept that some foods may be addictive has become popular in the recent years. Both animal and human models maintain far-reaching overlap between the neuronal signaling associated with consumption of delicious foods and substance addiction. On the other hand, some people are unable to stop eating particular foods in spite of their efforts to stop consuming these foods (Burke et al., 2016). They continue to eat these foods not quench their hunger, but to feel better emotionally. Even though food addiction may result in weight gain and overconsumption of calories, it is not the same as obesity (Burke et al., 2016; Ziaudden and Fletcher, 2012). The idea of food addiction is controversial. However, it has substantial effects …show more content…

Though, the majority of people consume unhealthy foods due to emotional reasons, but not as a result of the paucity of information. Food addiction appears plausible since the brain pathways that changed to react to natural rewards are triggered by addictive substances (Avena et al., 2008; Davis, 2013). Palatable foods such as sugar have the same characteristics as substances that produce dopamine and opioids. Hence, sugary foods may have addictive potential. The belief that some of the drugs may lead to addiction raises the probability that even some types of food result in food addiction. The study indicates that some people are forced to consume sweet foods in the same way alcoholic may feel forced to drink (Avena et al., 2008). Avena et al., research focused on sugar dependence evidence in a model of animal (Avena et al., 2008). The researchers analyzed four addiction components (bingeing, cross-sensitization, craving, and withdrawal). Further, they related these behaviors to neurochemical changes that take in the brain, and also occur with addictive substances such as alcohol. Neutral systems that progressed to stimulate and reinforce foraging and …show more content…

Avena et al., found that irregular availability of sugar to rats caused binge-like manner drinking. This stimulated the release of DA in the nucleus accumbens, the same as the typical influence of drug abuse. The release of DA in the nucleus accumbens results in alterations in the availability of extracellular dopamine receptor. Irregular availability of sugar to rats also acted like opioids in the brain (Avena et al., 2008; Baik et al., 2013). The researchers identified significant changes of opioid systems, for instance, declined enkephalin mRNA expression in the NAc(Avena et al., 2008). Therefore, the symptoms of withdrawal appear to be an influence of opioid changes because withdrawal can be achieved by antagonist naloxone of opioid. Surprisingly, food deprivation also has the ability to indicate opiate-like withdrawal symptoms (Avena et al., 2008).Withdrawal condition entails not less than two neurochemical manifestations. The first manifestation involves a decline in the extracellular DA in the NAc. Another manifestation entails the release of Ach (acetylcholine) from accumbens interneurons (Avena et al., 2008; Gearhardt et al., 2011).Adaptations of neurochemical in reaction to irregular sugar consumption imitate the influences of opiates(Dileone et al., 2016). It is postulated that irregular, large consumption

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